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Titlebook: Salt and Hypertension; Dietary Minerals, Vo R. Rettig,D. Ganten,F. C. Luft Conference proceedings 1989 Springer-Verlag Berlin Heidelberg 19

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Augmented Reciprocal α-2 and β-2 Adrenoceptor Changes and Rise of Total Peripheral Resistance Duringney cells so that a high salt intake would result in enhanced vasoconstriction, reduced vasodilatation, and enhanced sympathetically mediated proximal tubular sodium reabsorption in subjects genetically prone to hypertension. The above hemodynamic characteristics have been reported in patients with
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Role of Calcium in Modulating Salt Sensitivity (H. et al. 1984a,b) have noted no relationship between salt consumption and hypertension in the NHANESI population. Evidence is accumulating to indicate that suboptimal intakes of calcium, and possibly potassium, may predispose these populations to salt sensitivity. It has been noted (HARLAN et al.
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S. Oparil,Y.-F. Chen,R.-H. Yang,H. Jin,Q. C. Meng,E. J. Cragoe,J. M. Wyss
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P. J. Mulrow,E. Kusano,K. Baba,Y. Doi,D. Shier,R. Franco-Saenz
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M. H. Weinberger,F. C. Luft,J. Z. Miller,C. E. Grim,N. S. Fineberg,J. C. Christian
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