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Titlebook: Replacement of Renal Function by Dialysis; Drukker,Parsons,Maher,Walter H. Hörl,Karl M. Koch, Book 2004Latest edition Springer Science+Bus

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书目名称Replacement of Renal Function by Dialysis
编辑Drukker,Parsons,Maher,Walter H. Hörl,Karl M. Koch,
视频video
概述Includes supplementary material:
图书封面Titlebook: Replacement of Renal Function by Dialysis;  Drukker,Parsons,Maher,Walter H. Hörl,Karl M. Koch, Book 2004Latest edition Springer Science+Bus
描述The leading textbook on the subject. A completely rewritten and up-to-date fifth edition, based upon the highly respected fourth edition, edited by C. Jacobs, C.M. Kjellstrand, K.M. Koch and J.F. Winchester. .Considered the global resource for dialysis specialists, dialysis manufacturers and scientists for over two decades, this authoritative, highly acclaimed major reference work has been completely rewritten and revised in a much-awaited 5th edition. All previous chapters have been updated to include the very latest advancements and understandings in this critical and complex field. New sections include those on computerization of dialysis records, online monitoring and biofeedback, patient sexual function, patient selection and integration, use of exercise in improving patient health, design of randomized trials, and more. This new edition is truly global in scope and features the contributions the top experts from around the world.
出版日期Book 2004Latest edition
关键词Dialyse; anatomy; atherosclerosis; blood pressure; kidney; transplantation
版次5
doihttps://doi.org/10.1007/978-1-4020-2275-3
isbn_softcover978-94-015-7012-1
isbn_ebook978-1-4020-2275-3
copyrightSpringer Science+Business Media B.V. 2004
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Uremic toxicityhis results in a large number of symptoms, which are, however, variable from patient to patient and from moment to moment. Although the link between clinical deterioration and uremia was recognized many decades ago, our knowledge concerning the responsible factors remains incomplete. The quest for ‘
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The uremic syndrome and pathophysiology of chronic renal failureulation (1) (Figure 1). Atherosclerosis is a major risk factor in patients with chronic kidney diseases. The high prevalence of coronary heart disease and cardiac death in uremia is related to the accelerated atherosclerosis in these patients (2). A very rapid appearance of advanced coronary lesions
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Solute transport mechanisms in dialysisArtificial kidneys accomplish this by dialyzing and filtering the blood across semipermeable membranes, taking advantage of the natural forces of diffusion and convection. Native kidneys combine filtration at the glomerulus with selective reabsorption across the tubule. To allow movement of solute i
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Optimization of high-flux, hollow-fiber artificial kidneysaltering membrane permeability. This emphasis on membrane permeability was propelled largely by the middle molecule hypothesis and a desire to obtain solute removal characteristics for artificial kidneys similar to those of the native organ. As of a decade ago these achievements were approximately r
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The biology of hemodialysisa living animal may be submitted to dialysis outside the body, and again returned to the natural circulation without exposure to air, infection by microorganisms or any alteration which would necessarily be prejudicial to life (2). Their dialyzer had a series of celloidin tubes with a surface area o
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Vascular access for hemodialysisdstage renal disease (ESRD) patient via the blood stream permitting blood purification. Blood access is required at least three times a week with high blood flow (300–400 ml/min) to deliver a minimal ‘dialysis dose’. Patency of VA is of vital importance for the ESRD patient since it represents his/h
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