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Titlebook: Renal Disease: An Illustrated Guide; D. Gwyn Williams Book 1981 MTP Press Limited 1981 arteries.artery.biopsy.drugs.histology.immunology.k

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发表于 2025-3-21 17:50:01 | 显示全部楼层 |阅读模式
书目名称Renal Disease: An Illustrated Guide
编辑D. Gwyn Williams
视频videohttp://file.papertrans.cn/827/826993/826993.mp4
丛书名称Topics in Renal Disease
图书封面Titlebook: Renal Disease: An Illustrated Guide;  D. Gwyn Williams Book 1981 MTP Press Limited 1981 arteries.artery.biopsy.drugs.histology.immunology.k
描述This book does not pretend to be a comprehensive textbook of nephrology, nor is it a guide to the management of renal disease, as space does not allow the description or discus­ sion of investigation and treatment. Its intention is to illustrate the diverse diseases which present to nephrologists. Since the key to understanding the effects and progression of a disease are to know and com­ prehend its pathology, I have approached the subject not by discussing presenting symptoms of single diseases in different sections, which would lead to much repetition, but by describing the disorders according to the mechanisms which cause them. To illustrate the text I have chosen the most immediate clue which usually appears in a given dis­ ease, which may therefore be a clinical sign, a radiograph or the histological appearance of a renal biopsy. D. Gwyn Williams Guy‘s Hospital, London 1. Trauma Trauma is a common cause of acute renal failure. This may arise directly from injuries to the kidneys themselves despite their protected position (Figure 1). Trauma can also cause acute renal failure by damaging the renal arteries, which may be actually avulsed from the kidneys, or stretched at the ti
出版日期Book 1981
关键词arteries; artery; biopsy; drugs; histology; immunology; kidney; lead; management; metabolic disorder; nephrolo
版次1
doihttps://doi.org/10.1007/978-94-009-8066-2
isbn_softcover978-94-009-8068-6
isbn_ebook978-94-009-8066-2
copyrightMTP Press Limited 1981
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发表于 2025-3-21 20:43:57 | 显示全部楼层
Metabolic Disorders, of the causes of hypercalcaemia may be responsible, e.g. hyperparathyroidism, sarcoidosis, excess vitamin D, idiopathic hypercalcaemia of children and malignancy. Stone formation, however, does not occur in neoplastic hypercalcemia, as the metabolic abnormality is not sufficiently prolonged.
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Metabolic Disorders,tinctive radiological appearance (Figure 36)) or a reduction of kidney function and parenchymal loss without gross evidence of calcium deposition. Any of the causes of hypercalcaemia may be responsible, e.g. hyperparathyroidism, sarcoidosis, excess vitamin D, idiopathic hypercalcaemia of children an
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Obstruction,ic renal failure. Infection is a common complication. Other complications are stone formation, tubular dysfunction, hypertension and polycythaemia (rare). Symptoms range from none to pain, abdominal swelling due to an enlarged bladder, kidneys, or both, the symptoms of renal failure, and those direc
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not allow the description or discus­ sion of investigation and treatment. Its intention is to illustrate the diverse diseases which present to nephrologists. Since the key to understanding the effects and progression of a disease are to know and com­ prehend its pathology, I have approached the sub
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Book 1981 the description or discus­ sion of investigation and treatment. Its intention is to illustrate the diverse diseases which present to nephrologists. Since the key to understanding the effects and progression of a disease are to know and com­ prehend its pathology, I have approached the subject not b
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