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Titlebook: Renal Anemia; Conflicts and Contro Onyekachi Ifudu (Associate Professor of Medicine, Book 2002 Springer Science+Business Media Dordrecht 2

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书目名称Renal Anemia
副标题Conflicts and Contro
编辑Onyekachi Ifudu (Associate Professor of Medicine,
视频videohttp://file.papertrans.cn/827/826976/826976.mp4
图书封面Titlebook: Renal Anemia; Conflicts and Contro Onyekachi Ifudu (Associate Professor of Medicine,  Book 2002 Springer Science+Business Media Dordrecht 2
描述Is the nephrology community facilitating excess cardiovascular deaths in patients with kidney failure and anemia by treating to a subnormal hematocrit? Why have clinicians and nephrologists permitted health insurance companies and the government to decide when anemia therapy should begin in persons with progressive kidney failure? Is iron the only variable that can be manipulated to maximize response to recombinant erythropoietin? Are we using too much intravenous iron in kidney failure patients, and is oral iron supplementation worthless in sustaining iron stores during long-term erythropoietin treatment? When does left ventricular hypertrophy begin to emerge in patients with progressive renal disease and is there convincing evidence that anemia is a significant cause of LVH in this setting? Is darbepoetin alfa, a new novel, long-acting erythropoietin, really superior to recombinant erythropoietin? This book is a compilation of proceedings from a conference in Brooklyn convened to address these and other controversial and unresolved issues in renal anemia management.
出版日期Book 2002
关键词anemia; erythropoietin; kidney; nephrology; sepsis; diabetes
版次1
doihttps://doi.org/10.1007/978-94-015-9998-6
isbn_softcover978-90-481-6045-7
isbn_ebook978-94-015-9998-6
copyrightSpringer Science+Business Media Dordrecht 2002
The information of publication is updating

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Clinton D. Brown,Zhong H. Zhao,Lorraine L. Thomas,Robert deGroof,Eli A. Friedman
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Onyekachi Ifudu (Associate Professor of Medicine,
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Is long-term intravenous iron therapy risky?,Cs also contributed to iron overloading in the past and was associated with iron deposits in liver reticuloendothelial and parenchymal cells. However, iron overloading was not associated with functional changes in the liver (i.e., increases in liver function tests or cirrhosis) unless PRBC transfusions resulted in viral hepatitis [1].
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Normalizing hematocrit in renal failure: dangerous or desirable?, or fully corrected is arguably the most debated and controversial issue in this aspect of patient management. Why is this so? If one examines the scientific evidence relating to this topic, one can find both circumstantial evidence, and also evidence from intervention clinical trials [4–11].
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Epilogue,traordinary impact of treatment with erythropoietin in functionally anephric peritoneal dialysis and hemodialysis patients, Ifudu convened diverse professionals with shared interest in the uremic patient and concentrated their attention on the remarkable positive change in rehabilitation potential imparted by recombinant erythropoietin.
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