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Titlebook: Relaxin 2000; Proceedings of the T Geoffrey W. Tregear,Richard Ivell,John D. Wade Conference proceedings 2001 Springer Science+Business Med

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https://doi.org/10.1007/978-94-017-2877-5G proteins; Glycogen; Mamma; gene expression; growth factor; insulin; insulin-like; physiology; proteins; sig
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Relaxin is an important factor for uterine differentiation and implantation in the marmoset monkey birth canal and at least in some species a suppression of spontaneous contractions [1,2]. Such findings are generally observed in animal models, like the rat and pig, which show a marked relaxin physiology with maximum relaxin levels during the last trimester of pregnancy. For primates, however, this classic role of relaxin is open to question.
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Hooked on relaxin: A career-long love affair with a fickle and elusive hormonewhich was published with the conference proceedings [1]. Therefore, rather than go over the same ground again, . would like to review some recent studies that have reinforced my belief that relaxin remains an elusive — yet seductive — hormone for investigation. Your indulgence is then sought for som
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Relaxin retards apoptosis in the cervix and the vagina during pregnancy in ratshree fold [1–3], and vaginal wet weight increases about two fold [3]. Relaxin, which is secreted by the corpora lutea throughout the second half of rat pregnancy [5], plays a major role in promoting this growth of the lower reproductive tract. Relaxin induces more than a 40% increase in the wet weig
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Synthesis and function of relaxin in human endometrium is therefore necessary to suggest a role for relaxin in human endometrial function. Previous data demonstrate that relaxin is present in the endometrium during the late luteal phase. Immunohistochemical studies of endometrium from patients at different stages of the menstrual cycle demonstrated loc
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Analysis of relaxin deficient mice in the uterus, brain and prostate gland [2–4]. Binding sites for relaxin include uterus, cervix, mammary gland, nipple, pubic symphysis, cardiac atrium and brain [5–8]. Much of our knowledge of the biological actions of relaxin has been derived from studies in the rat. Removal of the source of circ
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