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Titlebook: Regulation of Gene Expression by Hormones; Kenneth W. McKerns Book 1983 Springer Science+Business Media New York 1983 DNA.Gonadotropin.Hyp

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书目名称Regulation of Gene Expression by Hormones
编辑Kenneth W. McKerns
视频video
丛书名称Biochemical Endocrinology
图书封面Titlebook: Regulation of Gene Expression by Hormones;  Kenneth W. McKerns Book 1983 Springer Science+Business Media New York 1983 DNA.Gonadotropin.Hyp
描述The International Foundation for Biochemical Endocrinology is incorporated as a nonprofit research and educational organization. It is dedicated to the dissemination of knowledge, cooperative research programs, and cultural interaction on an international basis. The Foundation is concerned with both basic research and practical applications of biological knowledge to the betterment of humanity. Among our interests are global resource management, human reproduction, hormonal regulation of normal and cancer cells, study of aging and degenerative diseases, brain peptides, peptide neurotransmitter compounds, mechanism of action of hormones, peptide hormone synthesis, and recombinant DNA techniques. This monograph is the ninth sponsored by the Foundation in the Biochemical Endocrinology series. The previous four have been: Hormonally Active Brain Peptides: Structure and Function (1982), K. W. McKerns and V. Pantie, eds.; Reproductive Processes and Contraception (1981), K. W. McKerns, ed.; Synthesis and Release of Adenohypophyseal Hormones (1980), M. Jutisz and K. W. McKerns, eds.; and Structure and Function of the Gonadotropins (1978), K. W. McKerns, ed. These have all been published by
出版日期Book 1983
关键词DNA; Gonadotropin; Hypophyse; gene expression; genes; hormone; reproduction; diabetes
版次1
doihttps://doi.org/10.1007/978-1-4684-4418-6
isbn_softcover978-1-4684-4420-9
isbn_ebook978-1-4684-4418-6
copyrightSpringer Science+Business Media New York 1983
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https://doi.org/10.1007/978-1-4684-4418-6DNA; Gonadotropin; Hypophyse; gene expression; genes; hormone; reproduction; diabetes
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Estrogen-Induced Uterine Hypertrophy,on of existing ribosomes and the estrogen-induced synthesis of new ribosomes. This review summarizes our current knowledge of the events leading up to, and apparently prerequisite for, this activation process.
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Modulation of an Estrogen-Induced Protein in the MCF7 Human Mammary Cancer Cell,the biosynthesis of specific proteins. The mechanism of the pleiotypic effect is poorly understood and may involve common intermediary steps between the binding of the receptor in the nucleus and the final responses. In any case, this pleiotypic effect can also be triggered by other hormones and is therefore not specific for estrogens.
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Hormone Action in Human Breast Cancer Estrogen Regulation of Specific Proteins,rted in premenopausal patients following oophorectomy (Beatson, 1896). However, systematic exploitation of endocrine ablative surgery did not occur until the 1950s when Huggins and Dao (1954) obtained 38 clinical remissions after performing 100 ovariectomies for palliation of advanced breast cancer.
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Stimulation of Milk-Fat Synthesis in Mammary Epithelioid Cells by Progesterone,ent from that occurring in adipose tissue. The pentose phosphate pathway predominates and glucose-6-phosphate dehydrogenase is much more sensitive to insulin (Leader and Barry, 1969; Oka .., 1974). In adipose tissue glucose, lactate, and pyruvate serve equally well as precursors of fatty acids, but
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Prolactin and Casein Gene Expression in the Mammary Cell,egnancy, the mammary gland is restricted to a few duct cells. During pregnancy, under the influence of estrogens, progesterone, and growth factors, the secretory cells progressively appear. They are organized in an epithelium forming a large number of alveoli. At the end of pregnancy, many alveolar
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