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Titlebook: Recognition and Response in Plant-Virus Interactions; Ron S. S. Fraser (Workshop Director) Conference proceedings 1990 Springer-Verlag Ber

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书目名称Recognition and Response in Plant-Virus Interactions
编辑Ron S. S. Fraser (Workshop Director)
视频video
丛书名称Nato ASI Subseries H:
图书封面Titlebook: Recognition and Response in Plant-Virus Interactions;  Ron S. S. Fraser (Workshop Director) Conference proceedings 1990 Springer-Verlag Ber
描述Mechanisms of resistance to plant viruses are diverse, and probably involve different types of recognition events. Often, a cascade of changes affecting broader aspects of defence and metabolism is switched on progressively after the initial recognition event. Virulence, i.e. resistence-breaking behaviour of the virus, involves a failure or alteration of recognition or subsequent signalling. Consequences of these recognition events are the ways in which the pathogenic effects on the host are exerted: formation of visible symptoms and control of plant growth. This volume offers a comprehensive coverage of the recognition and signalling events between plants and viruses whereby the particular attraction of viruses (and viroids) is that they can now be completely defined in molecular terms: they offer excellent opportunities for studying the molecular biology of signalling, and may even provide useful guidelines on how plants and cellular pathogens interact.
出版日期Conference proceedings 1990
关键词Molekularbiologie; Pathogen; Resistenz; Signalmoleküle; Virus; Viruses; metabolism; virulence
版次1
doihttps://doi.org/10.1007/978-3-642-74164-7
isbn_softcover978-3-642-74166-1
isbn_ebook978-3-642-74164-7Series ISSN 1010-8793
issn_series 1010-8793
copyrightSpringer-Verlag Berlin Heidelberg 1990
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Specificity and Recognition Events in the Transmission of Plant Viruses by Vectorse highly specific. Vector specificity is determined by recognition events between virus proteins and sites of retention in the vector. Virus particle protein is invariably involved but in some instances recognition is mediated by a non-particle protein (helper component), or by the particle protein
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Plant Virus Epidemiology: The Battle of the Genes All three components of the pathosystem are subject to genetic variability and there is abundant evidence of the way in which virus strain, host cultivar and vector biotype can influence the onset and course of epidemics. Indeed, virus epidemiology can be considered as a “battle” of virus, host and
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Signals and Structures Involved in Early Interactions between Plants and Viruses or Pseudovirusesexposed portion of RNA. No cell specificity exists during virus uptake or early cotranslational disassembly. Virus-like reporter ribonucleocapsids were synthesized . to study the mechanism, sequence-dependence and site of disassembly and transient gene expression without concomitant replication. “Ps
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Genetic Organization, Evolution and Expression of Plant Viral RNA Genomesmay have either segmented or nonsegmented genomes that are expressed using a variety of translation strategies. Despite this variation, nucleotide sequence data reveal many interviral relationships and indicate that plant RNA viruses can be grouped into a small number of clusters. A review will be g
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Replication of Plant RNA Viruses , in Relation to Virus-Host Interactionsrtments due to virus infection can indicate the parts of the cell likely to be involved in replication. The sites at which RNA replication occurs can also be ascertained by investigating where virus-specific, newly synthesized or double-stranded RNA species are located, or where the viral nonstructu
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Viroid Replication Mechanisms, circular RNA molecule of 246–375 nucleotides. The autonomous replication of viroid RNA in the infected cell requires a close interaction with host factors. At least two replication mechanisms for different viroids can be envisaged. This chapter gives a brief review of possible mechanisms of viroid
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