Titlebook: Recent Advances in Thrombosis and Hemostasis; Kenzo Tanaka (Professor Emeritus),Earl W. Davie (P Book 2008 Springer-Verlag Tokyo 2008 Card

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Positive and Negative Regulation of Integrin FunctionS-1 have been identified as negative regulators. These molecules are secreted from or expressed on endothelial cells and inhibit the function of platelets as well as α.β.. Investigation on these positive and negative regulatory factors should provide a new insight into the treatment of pathological thrombosis.

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Book 2008ertension, and obesity are the leading causes of morbidity and mortality. They have become urgent medical problems with serious economic consequences in industrialized and devel- ing countries alike. At the same time, the impact of molecular biology and genetics on our understanding of thrombosis an

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lic disorders as well as related diseases such as malignancies, arteriosclerosis, diabetes mellitus, hypertension, and obesity are the leading causes of morbidity and mortality. They have become urgent medical problems with serious economic consequences in industrialized and devel- ing countries ali

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移植

High-Mobility Group Box 1: Missing Link Between Thrombosis and Inflammation?elial cells, plays an important role in sequestering HMGB1. TM may prevent HMGB1 from reaching remote organs, thereby restricting the range of HMGB1 action to the site of injury. In this chapter, we review recent progress made in defining the physiological and pathological roles of HMGB1 and therapeutic strategies aimed at blocking HMGB1.

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Structure and Function of the Endothelial Cell Protein C Receptorficity of APC and enables proteolysis of PAR-1, leading to intracellular signal transduction. The EPCR-APC-PAR-1 signaling induces various genes and explains the antiinflammatory, antiapoptotic, and wound healing effects of APC.

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