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Titlebook: Oxygen Transport to Tissue XXXI; Eiji Takahashi,Duane F. Bruley Conference proceedings 2010 The Editor(s) (if applicable) and The Author(s

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The Effects of Cilostazol on Tissue Oxygenation upon an Ischemic-reperfusion Injury in the Mouse Cerd intraperitoneally 1 h before sacrifice and capillary patency was assessed by fluorescein isothiocyanate-labeled Lycopersicon esculentum lectin bound to the endothelial surface. Treatment with cilostazol markedly increased the capillary patency which was accompanied by a reduction in the hypoxic ar
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T-state Stabilization of Hemoglobin by Nitric Oxide to Form α-Nitrosyl Heme Causes Constitutive Releide with activation of glycolysis of the cells, so far as judged by the fact that intracellular ATP was significantly decreased by the NO treatment. Collectively, the present study suggests that hypoxia-induced ATP release is triggered through mechanisms involving R-T transition of Hb, and the gatin
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