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Titlebook: Oxidative Stress and Neuroprotection; H. Parvez,P. Riederer Book 2006 Springer-Verlag Vienna 2006 Alzheimer.Morbus Parkinson.Parkinson.alz

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书目名称Oxidative Stress and Neuroprotection
编辑H. Parvez,P. Riederer
视频video
丛书名称Journal of Neural Transmission. Supplementa
图书封面Titlebook: Oxidative Stress and Neuroprotection;  H. Parvez,P. Riederer Book 2006 Springer-Verlag Vienna 2006 Alzheimer.Morbus Parkinson.Parkinson.alz
描述.This book deals with basic and clinical aspects of monoamine oxidase (MAO) subtypes A and B highlighting its importance in neurological and psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson’s disease (PK) and depression are the focus of several chapters. As MAO is the basis of the development of the "oxidative stress hypothesis" of PD, several chapters are devoted to iron and iron-induced oxidative stress in various experimental studies and clinical conditions. Based on these findings, new compounds have been developed which not only block MAO, but are in addition, either inhibitors of acetylcholine esterase or have iron chelating properties. The aspect of "preclinical" and "clinical" neuro protection as well as MAO neuroprotection are additional topics covered in this book. MAO, iron and neuroprotection are seen in the framework of general anti Parkinson’s therapy with chapters on levodopa, dopaminergic receptor agonists and clinical issues. .
出版日期Book 2006
关键词Alzheimer; Morbus Parkinson; Parkinson; alzheimer‘s disease; apoptosis; brain; cell; cytokine; dementia; depr
版次1
doihttps://doi.org/10.1007/978-3-211-33328-0
isbn_softcover978-3-7091-1736-1
isbn_ebook978-3-211-33328-0Series ISSN 0303-6995
issn_series 0303-6995
copyrightSpringer-Verlag Vienna 2006
The information of publication is updating

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Acute and chronic effects of developmental iron deficiency on mRNA expression patterns in the braindevelopment in a metabolically compromised setting that given appropriate intervention is mostly correctable. There are, however, long term consequences to the developmental iron deficiency that could underlie the neurological deficits reported for iron deficiency.
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0303-6995 psychiatric diseases. Consequently the therapeutic actions of MAO-A and -B inhibitors in Parkinson’s disease (PK) and depression are the focus of several chapters. As MAO is the basis of the development of the "oxidative stress hypothesis" of PD, several chapters are devoted to iron and iron-induce
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,The DONPAD-study — Treatment of dementia in patients with Parkinson’s disease with donepezil,dizziness and confusion. This may result from the titration regime of donepezil, that allows only 5 and 10mg dosages. Participants with premature study termination had a significant longer duration of PD, less motivation and sleep disturbances at night. Treatment with donepezil was only effective in
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