Titlebook: Oncogenes and Tumor Suppressor Genes in Human Malignancies; Christopher C. Benz,Edison T. Liu Book 1993 Kluwer Academic Publishers 1993 DN

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Involvement of G proteins, cytoplasmic calcium, phospholipases, phospholipid-derived second messengat modulates the transcription of numerous genes that contain AP-1 control elements. The activation of AP-1(Fos:Jun) in response to mitogenic stimuli occurs during the transition between the G. phase (the resting stage of the cell cycle) and G., phase (the stage when the cell becomes committed to un

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DNA binding by the Myc oncoproteins, of c-. [.]. In contrast to many other oncogenes that have activating mutations in the coding sequence, this deregulated expression of a normal c-. coding sequence appears to be responsible for the oncogenic contribution of c-. [.].

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Angiogenesis: A mechanism by which oncogenes and tumor suppressor genes regulate tumorigenesis,-oncogenic tumor suppressor genes) increases the likelihood that a cell will become tumorigenic. A complete understanding of how the activation of an oncogene contributes to tumorigenicity demands additional information. It requires (1) that the oncogene-regulated genes be identified and the functio

Ingredient

Book 1993loss-of-function genetic abnormalities, as well as autocrine and paracrine growth factor loops known to regulate tumor physiology and malignant cell behavior. Curiously, many of these genetic and functional abnormalities are shared by several different tumor types and are not uniformly present in al

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0927-3042 ant cell behavior. Curiously, many of these genetic and functional abnormalities are shared by several different tumor types and are not uniformly present in al978-1-4613-6349-1978-1-4615-3088-6Series ISSN 0927-3042 Series E-ISSN 2509-8497

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