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Titlebook: Nutritional Intervention in the Aging Process; H. James Armbrecht,John M. Prendergast,Rodney M. C Book 1984 Springer-Verlag New York, Inc.

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Changes in Calcium and Vitamin D Metabolism with Ageteoporosis are multiple in nature and are still the object of intense investigation (1). However, it is known that osteoporosis is aggravated by two factors: (a) lack of calcium in the diet and (b) poor absorption of dietary calcium by the intestine (2,3).
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Dietary Restriction and Biologic Variablesen reported in the following model systems: . sp., rotifers, . sp., and fish. In addition, a number of laboratory experiments have been carried out on rodents. It has been generally believed that nutritional manipulations that increase life span had to be imposed during early growth. This concept or
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Aging in the Food-restricted Rat: Body Temperature, Receptor Function, and Morphologic Changes in thand old adults, receptor sensitivity in young and old adults, and neuromorphologic changes in extreme old age. We were interested to see 1) whether lower body temperature was a factor in the longevity of food-restricted rats, 2) whether receptor function was one of the age-sensitive variables affect
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Dietary Antioxidants, Membrane Lipids, and Aging is the peroxidative attack of the oxygen-containing radicals on membrane lipids. In fact, free radical-induced lipid peroxidation has become an important basis for explaining the biochemical phenomenon of cellular aging. Many types of organ damage can be directly or indirectly traced to an increase
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Membrane Changes Associated with Alcohol Use and Aging: Possibilities for Nutritional Interventionng effects. Unfortunately, a significant number of individuals drink large amounts of alcohol, which can result in a wide range of physical, psychologic, and social problems. In the past few years, increasing attention has been focused on alcohol consumption by elderly individuals (2–4). While the e
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Calcium Supplementation and Osteoporosishat of younger, or menstruating, individuals although the remaining bone is normal in chemical composition. Symptomatic senile or postmenopausal osteoporosis syndrome was classically considered to result from the universal loss of bone that normally attends senescence in both sexes and begins in the
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