Titlebook: Nitric Oxide, Cytochromes P450, and Sexual Steroid Hormones; Jack R. Lancaster,John F. Parkinson Conference proceedings 1997 Springer-Verl

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D. V. Fallerstem, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease

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H. W. Schnaper,K. A. McGowan,S. C. Hubchak,M. C. Cid,H. K. Kleinman,S. Kim-Schulzestem, and hypoxia, such as the PHD-HIF system, have recently been explored in various cells, including kidney cells. These mechanisms include intracellular sensors for oxidative stress and hypoxia. This means that novel approaches targeting these sensors may offer clinical benefits in kidney disease

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Nitric Oxide, Cytochromes P450, and Sexual Steroid Hormones

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Nitric Oxide and Cyclooxygenases, NO and PGs contributes to the antiinflammatory properties of NOS inhibitors. I will present evidence that COX enzymes are targets for the pathophysiological roles of NO and that, once activated in the presence of NO, these represent important transduction mechanisms for the actions of NO.

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Regulation of Endothelial Nitric Oxide Synthase by Estrogen,nt of NO synthesis may therefore inhibit atherogenesis. NO is generated by the nitric oxide synthase (NOS) family of proteins referred to as neuronal (nNOS, NOS 1), inducible (iNOS, NOS 2), and endothelial NOS (eNOS, NOS 3) (Sessa 1994). These enzymes are distinct gene products and they catalyze the

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Nitric Oxide Deficiency in Preeclampsia,ate that steroid hormones, particularly progesterone, are involved in the regulation of maternal adaptation of the cardiovascular system during pregnancy. Furthermore, our findings also demonstrate that progesterone is the main factor regulating the L-arginine-NO-cGMP system (NO system) in the uteru