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Titlebook: New Trends in Allergy III; Johannes Ring,Bernhard Przybilla Conference proceedings 1991 Springer-Verlag Berlin Heidelberg 1991 Allergie.al

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Epidemiology of Contact Allergyto the source population studied. A prospective study in Heidelberg, West Germany, in which 1155 patients were examined during a 2 year period (1986–1988) provided these results: (1) women outnumber men by 2:1; (2) the hands and face are primarily affected in young women, in older patients the legs
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Cloning of Birch Pollen Allergenspores of molds, and dander from domestic animals. Pollen of the white birch (.) is one of the main causes of IgE-mediated reactions in allergic patients. Bet v I, an allergenic protein with a molecular mass of 17 kDa, is a constituent of white birch pollen and is responsible for IgE binding in more
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Possible Approaches to Controlling the IgE Antibody Responsecess. Indeed, anti-IL-4 and IFN. inhibited the IgE formation both in vitro and in vivo. However, the quantity of the antibody or IFN. required for the suppression of the in vivo IgE synthesis is too much for clinical applications. Another approach for the suppression of IgE antibody response is anti
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Human Fc,RII/CD23 in the Regulation of the Allergic Immune Responseotein of 45 kDa which is constitutively expressed on resting, .... B lymphocytes and is lost after isotype switch. Fc.RII/CD23 is also found on follicular dendritic cells, eosinophils, platelets, alveolar macrophages and can be induced on monocytes and Langerhans cells by IL-4. Two isoforms (CD23a a
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