Titlebook: New Strategies in Stroke Intervention; Ionic Transporters, Lucio Annunziato Book 2010 Humana Press 2010 brain damage.ionic homeostasis.isc

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Why have Ionotropic and Metabotropic Glutamate Antagonists Failed in Stroke Therapy?,tamate were destined to die Later on, in 1985, glutamate toxicity was associated with anoxic cell death, since anoxic depolarization resulted in the release of glutamate into the extracellular compartments Similarly, in 1987, Choi indicated that glutamate was a remarkably potent and rapidly acting n

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Endoplasmic Reticulum Calcium Homeostasis and Neuronal Pathophysiology of Stroke,in neurones and in glia Morphologically, the ER is represented by a system of microtubules and cisternae organised into a complex 3D network. In neurones, the ER extends from the nucleus and the soma to the dendritic arborisation and, through the axon, to presynaptic terminals. Although the lumen of

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,,,,,,llular H. for extracellular Na. and in doing so regulates intracellular pH (pH.) homeostasis and cell volume. NHEs contain two functional domains: the N-terminal transmembrane domain, which is necessary and sufficient to catalyze ion translocation, and the C-terminal cytoplasmic domain, which is cru

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Role of TRPM7 in Ischemic CNS Injury,e and consequent overactivation of NMDARs occurs rapidly (time span of minutes to hours), the ensuing neuronal death has been noted to progress with some delay, the phenomena being appropriately referred to as delayed neuronal death (DND). Surprisingly, given the massive release of glutamate and str