Titlebook: New Horizons for Failing Heart Syndrome; Shigetake Sasayama Book 1996 Springer Japan 1996 Failing Heart Syndrome.circulation.heart.heart f

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湿润

Akira Matsumori,Takehiko Yamada,Shigeo Matsui,Tetsuo Shioi,Shigetake Sasayamalates. The characterization of face/core debonding is a major topic of this text, and analysis methods based on fracture mechanics are described and applied to several contemporary test specimens. Test methods and results documented in the literature are included and discussed. The book will benefit

CESS

Molecular Aspects of Mechanical Stress-Induced Cardiac Hypertrophy and Failureing that LVH is not a simple adaptation to an increased external load. Many investigators have extensively studied the activation of a novel program of gene expression in the heart induced by pressure overload (i.e., the transient induction of immediate early genes and the reexpression of fetal-type

钢笔尖

Calcium-Activated Neutral Proteases and Myocardial Protein Catabolismells. Its basic aspects have been clarified regarding biochemical profile, autoactivation, isoforms, subunit structure, functional domain, gene structure, and amino acid sequence deduced from cDNA. Its in vitro characteristics and interaction with endogenous inhibitor (calpastatin) specific to CANP

远足

achlorhydria

Two Major Pumps of the Heart: Difference and Interdependence of the Right and Left Ventriclesentricles propel the same amount of blood but are different with regard to intracavitary pressure, wall thickness, and shape. Coronary perfusion pressure and regional wall motion are closely linked to myocardial metabolic states and regional myocardial blood flow. When coronary perfusion pressure is

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寡头政治

Mechanoenergetics of Acute Failing Hearts Characterized by Oxygen Costs of Mechanical Energy and Cons proved to be relatively constant under various physiological conditions but have been shown to be altered under pathophysiological conditions such as myocardial stunning and acidosis. In the stunned and acidotic hearts, the 0. costs of Emax were higher than in the normal hearts without marked chan