Titlebook: Neurotransmitters and Cortical Function; From Molecules to Mi Massimo Avoli,Tomás A. Reader,Pierre Gloor Book 1988 Springer Science+Busines

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Functional Organization of Glutamatergic Synapses,w attention to this “nonessential” amino acid by suggesting its importance in cerebral metabolism (Quastel and Wheatley, 1932). In the ensuing years, the neurochemical importance of glutamate was traced mainly to several metabolic processes: (1) oxidative deamination to α-oxoglutarate, (2) transamin

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Anatomy of Putative Glutamatergic Neurons,on of glutamate in the tissue and for discriminating the putative transmitter role of glutamate from its role in general cellular metabolism. The various markers useful for studying the localization, structure, and connections of neurons likely to transmit their message by releasing glutamate are th

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,-Glutamate and Its AgonistsSynaptic and Ionic Mechanisms in the Central Nervous System,dered as neurotransmitter candidates in the vertebrate CNS. Both Glu and Asp are present in large quantities in brain nervous tissue (about 10 and 2 mM, respectively) (Berl and Waelsch, 1958; Perry e.., 1981; Schousboe .., 1975); Glu is released from several cortical areas following stimulation of a

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GABA Neurons and Their Cotransmitters in the Primate Cerebral Cortex,ent in the mammalian cerebral cortex for a relatively long time (Awapara .., 1950; Roberts and Fenkel, 1950; Albers and Brady, 1959). Along with glutamate, GABA is the compound released in largest amounts when the cortex is subjected to depolarizing stimuli (Jasper and Koyama, 1969; Baughman and Gil

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GABAergic Processes in the Central Visual System,gical characteristics of the GABAergic cells (Iversen .., 1971; Sillito, 1975a, 1984; Ribak, 1978; Peters and Fairén, 1978; Peters and Regidor, 1981; Somogyi .., 1981, 1984; Freund .., 1983). My purpose in this account is to provide an overview of the range and types of contributions that GABAergic

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GABAergic Mechanisms and Epileptic Discharges,used by abnormal synchronous and excessive discharges of large neuronal populations (Taylor, 1931; Gastaut and Broughton, 1973; Avoli and Gloor, 1987). Based on the fact that normal function in the brain is dependent upon a balance between excitatory and inhibitory processes in neuronal circuits, tw