Titlebook: Neuroplasticity; New biochemical mech J. A. Costa e Silva,J. P. Macher,J. P. Olié Book 2009 Springer Healthcare Ltd. 2009 AMPA receptor.Cel

参考书目

https://doi.org/10.1007/978-1-908517-18-0AMPA receptor; Cellular plasticity; Glutamatergic modulation; depression; lead; mood disorder; pathophysio

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nurture

fatty-acids

concluding chapters are devoted to topics such as abdominal trauma, spiral CT in children, and CT-guided interventional procedures.978-3-540-42291-4978-3-642-56976-0Series ISSN 0942-5373 Series E-ISSN 2197-4187

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,Neuroplasticity — A New Approach to the Pathophysiology of Depression,ic antidepressants (TCAs) and the monoamine oxidase inhibitors, that have now been used for more than half a century. These compounds have been shown to overcome deficits in serotonin, noradrenaline and possibly dopamine function in the brain. This finding formed the basis for the monoamine hypothes

淡紫色花

Glutamate-Mediated Neuroplasticity Deficits in Mood Disorders,iency require neuronal adaptations involving a number of brain regions and neurotransmitter systems. Neuroplasticity is a broad term referring to a collection of events critical for neuronal adaptation, including those that occur at the molecular, cellular and systemic levels. While a number of diff

Carminative

Regulation of Cellular Plasticity in Mood Disorders: The Role of the AMPA Receptor,on conditions remain unclear. For much of the second half of the 20th century, the principal focus was on the role of monoamine neurotransmitters (in particular noradrenaline and serotonin, as well as dopamine) and their receptors, which are prominent in the limbic regions and prefrontal cortical ar

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Clinical Consequences of the Role of Glutamate and Neuroplasticity in Depressive Disorder,er, through its impact on neuroplasticity. However, a significant part of available information is based on animal studies, preclinical works and clinical research, and therefore this hypothesis has to be challenged at a broader clinical level.

receptors