Titlebook: Neuropharmacology of Ethanol; New Approaches Roger E. Meyer,Michael J. Lewis,Steven M. Paul Book 1991 Springer Science+Business Media New Y

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Alcohol and Anesthetic Actions: Are They Mediated by Lipid or Protein?,ss of drug and general anesthetics. Thus, a recent textbook of pharmacology summarizes the matter as “Despite popular belief in its stimulant properties, ethanol is entirely depressant in its actions on neurones of the central nervous system. In fact, its actions are qualitatively similar to those o

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,Probing Molecular Sites of Action for Alcohol’s Acute and Chronic Effects on Synaptoneurosome Membrroduces tolerance and physical dependence. The study of alcohol’s effects on the CNS has been complicated by the absence of a specific binding site that would indicate the primary locus of action for this molecule on the cells of the CNS. Unlike other drugs of abuse such as opiates, psychomotor stim

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Ethanol and the GABAA Receptor-Gated Chloride Ion Channel,tions of ethanol are not well understood. Ethanol has many of the physiochemical properties of other anesthetic agents, conferring non-specific disordering effects on neuronal membranes, especially at high concentrations. It is generally acknowledged that ethanol shares more specific pharmacologic a

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Ethanol and the NMDA Receptor: Insights into Ethanol Pharmacology,tlieb, 1926). Since then it has been shown that alcohols can partition into cell membranes and perturb the structure of the bulk membrane lipids (Chin and Goldstein, 1977), and that the intoxicating potency of alcohols is positively correlated with their lipid-perturbing capacity (Lyon et al., 1981)

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