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Titlebook: Neuropathogenic Viruses and Immunity; Steven Specter,Mauro Bendinelli,Herman Friedman Book 1992 Plenum Press, New York 1992 immune system.

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书目名称Neuropathogenic Viruses and Immunity
编辑Steven Specter,Mauro Bendinelli,Herman Friedman
视频video
丛书名称Infectious Agents and Pathogenesis
图书封面Titlebook: Neuropathogenic Viruses and Immunity;  Steven Specter,Mauro Bendinelli,Herman Friedman Book 1992 Plenum Press, New York 1992 immune system.
描述There has been a tremendous increase in interest in the neuropathogenicity of viruses during the past decade as we have come to recognize that the human immunodeficiency virus, which causes the acquired immunodeficiency syndrome (AIDS), can infect glial cells and cause neurological disease. Yet this increase has not been limited to AIDS but has extended to viruses that infect either or both the central and peripheral nervous systems. The changes examined here include both neurological and psychological diseases or syndromes. Moreover, the chapters in this volume review the interaction of the host immune system with the viruses examined and how such interactions may increase or decrease the neuropatho­ genicity of the viruses. Questions regarding viral neuropathogenesis include: (I) What is the mode of transmission of virus to the nervous system? (2) What types of cells are infected, and do they contain receptors for the virus? (3) What is the extent of damage that results from viral infection? (4) What are the immunologic mecha­ nisms by which damage is mediated or limited? Many of these questions remain unanswered, but this volume delves into efforts to provide some answers.
出版日期Book 1992
关键词immune system; immunodeficiency; infection; infections; nervous system; system; virus
版次1
doihttps://doi.org/10.1007/978-1-4684-5886-2
isbn_softcover978-1-4684-5888-6
isbn_ebook978-1-4684-5886-2Series ISSN 1075-1289
issn_series 1075-1289
copyrightPlenum Press, New York 1992
The information of publication is updating

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Immune Responses and the Central Nervous Systemechanism for antibody production, no lymphatic system, and few if any phagocytic cells. The CNS has been described as an “immunologically privileged site” because of the paucity of normal immune surveillance. Consequently, when a virus penetrates the blood—brain barriers that exclude most infectious
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Visna, a Lentiviral Disease of SheepKarakul sheep in 1933.. Almost three decades elapsed until the causative agent of visna, a virus, was isolated from the central nervous system (CNS) of visnaaffected sheep.. A few years later a virus was isolated from the lungs of a sheep with maedi.. The viruses were shown to be serologically relat
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Theiler’s Virus-Induced Demyelinating Disease in Mice frequently referred to as Theiler’s murine encephalomyelitis viruses (TMEV).. Their host range appears to be quite narrow, and serological evidence indicates that . is the natural host. The TMEV are present in virtually all nonbarrier mouse colonies throughout the world, where they cause asymptomat
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Neurotropic Retroviruses of Mice, Cats, Macaques, and Humans(HTLV) have focused attention on the neurotropic properties of the family Retroviridae. This family is divided into three subfamilies, the oncoviruses, the lentiviruses, and the spumaviruses, each of which is known to infect the central nervous system (CNS) of their natural animal hosts.. The only k
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Scrapiecluding visna and scrapie.. In comparing his results with those obtained in the burgeoning field of virology, Sigurdsson proposed a new category of infectious diseases, slow infections. He proposed three criteria for slow infections: (1) a very long incubation period lasting from several months to m
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Pathogenesis and Immunology of Herpesvirus Infections of the Nervous Systemonly a small host range in which to maintain an infection: typically 10.–10. individuals are sufficient to maintain a varicella-zoster virus (VZV) infection in the population, compared with >10. individuals for measles virus.. The great success of the herpesviruses is attributed to a remarkable stra
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