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Titlebook: Neuroinflammation; Mechanisms and Manag Paul L. Wood Book 2003Latest edition Springer Science+Business Media New York 2003 Alzheimer.Parkin

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发表于 2025-3-21 18:37:14 | 显示全部楼层 |阅读模式
书目名称Neuroinflammation
副标题Mechanisms and Manag
编辑Paul L. Wood
视频video
概述Includes supplementary material:
丛书名称Contemporary Neuroscience
图书封面Titlebook: Neuroinflammation; Mechanisms and Manag Paul L. Wood Book 2003Latest edition Springer Science+Business Media New York 2003 Alzheimer.Parkin
描述In this thoroughly updated and revised edition of his much praised book, Paul L. Wood and a panel of leading researchers capture these new developments in a masterful synthesis of what is known today about the inflammatory mediators and cells involved in neurodegenerative diseases. This second edition contains extensive updates on the mediators produced by microglia and their role in neuroinflammatory-induced neuronal lysis. There is also increased coverage of the animal models used in the study of neuroinflammatory mechanisms, of the new imaging methods that allow the noninvasive evaluation of microglial activation in human neurodegernerative disorders, and of the role of neuroinflammation in amyloid-dependent neuronal lysis.
出版日期Book 2003Latest edition
关键词Alzheimer; Parkinson; alzheimer‘s disease; brain; imaging techniques; receptor; spinal cord; stroke
版次2
doihttps://doi.org/10.1007/978-1-59259-297-5
isbn_softcover978-1-4684-9720-5
isbn_ebook978-1-59259-297-5
copyrightSpringer Science+Business Media New York 2003
The information of publication is updating

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书目名称Neuroinflammation网络公开度




书目名称Neuroinflammation网络公开度学科排名




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Neurotoxic Mechanisms of Nitric Oxider ., exerting physiological roles in many organ systems (e.g., musculature, pulmonary, gastrointestinal, immune, renal, endocrinological, and reproductive systems) ., in addition to modulation of regional and systemic circulation .. NO is also involved in the developmental aspects of neurosynaptic plasticity ..
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g researchers capture these new developments in a masterful synthesis of what is known today about the inflammatory mediators and cells involved in neurodegenerative diseases. This second edition contains extensive updates on the mediators produced by microglia and their role in neuroinflammatory-in
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Chronic Intracerebral LPS as a Model of Neuroinflammationmmation causes AD. Rather, this chapter will outline the details of an animal model that has provided compelling evidence that inflammation develops in response to existing genetically determined conditions within brains of AD patients.
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Book 2003Latest editioned coverage of the animal models used in the study of neuroinflammatory mechanisms, of the new imaging methods that allow the noninvasive evaluation of microglial activation in human neurodegernerative disorders, and of the role of neuroinflammation in amyloid-dependent neuronal lysis.
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Neuroinflammation as an Important Pathogenic Mechanism in Spinal Cord Injuryss. Although the mechanisms involved in the secondary injury process are not fully understood, the activated leukocyte-induced vascular damage leading to neuroinflammation has been postulated to be one of the important pathomechanisms of acute spinal cord injury ..
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