Titlebook: Neuroglia in the Aging Brain; Jean S. Vellis Book 2002 Springer Science+Business Media New York 2002 Alzheimer.ELIZA.Parkinson.age.aging.a

放弃

Neurotoxic Injury and Astrocytescept of the German pathologist Virchow ., who postulated that neurons were embedded in a connective tissue to which he coined the name neuroglia, or nerve glue. Although erroneous, it has persisted as the preferred and generic term for these cells, or in its shortened form—“Glia.”

渗透

Ammonium Ion Transport in Astrocytesimated intracellular pH of 7.1 in brain, as compared with 7.4 in blood, accounts for a twofold excess of NH.+ in the brain. This distribution of NH.+ approximates the distribution of total ammonia because more than 98% of ammonia exists as NH.+ at pH 7.4 or below.

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esthetician

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uron-glia intercommunication. The contributors show how glial signals may be modulated by hormones, growth factors, neurotransmitters, intracellular metabolism, and intercellular exchanges, as well as by aging of the blood-brain barrier.978-1-61737-088-5978-1-59259-105-3

小臼

Book 2002he normal aging process and in the major disorders of old age. Topics range from the cellular and molecular changes that occur with aging-especially aging-associated activation of astrocytes and microglia and its relation to neuronal injury and repair-to neuron-glia intercommunication. The contribut

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Glia and Extracellular Space Diffusion Parameters in the Injured and Aging Brain and glial cell functions. Populations of neurons can interact both by synapses and by the diffusion of ions and neurotransmitters in the ECS Since glial cells do not have synapses, their communication with neurons is mediated by the diffusion of ions and neuroactive substances in the ECS.

perpetual

Book 2002ging-associated activation of astrocytes and microglia and its relation to neuronal injury and repair-to neuron-glia intercommunication. The contributors show how glial signals may be modulated by hormones, growth factors, neurotransmitters, intracellular metabolism, and intercellular exchanges, as well as by aging of the blood-brain barrier.

insidious

of and changes in glial cells both during the normal aging process and in the major disorders of old age. Topics range from the cellular and molecular changes that occur with aging-especially aging-associated activation of astrocytes and microglia and its relation to neuronal injury and repair-to ne