Titlebook: Neurodegenerative Diseases; Molecular and Cellul Gary Fiskum Book 1996 Springer Science+Business Media New York 1996 Alzheimer.Amino acid.C

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书目名称Neurodegenerative Diseases影响因子(影响力)




书目名称Neurodegenerative Diseases影响因子(影响力)学科排名




书目名称Neurodegenerative Diseases网络公开度




书目名称Neurodegenerative Diseases网络公开度学科排名




书目名称Neurodegenerative Diseases被引频次




书目名称Neurodegenerative Diseases被引频次学科排名




书目名称Neurodegenerative Diseases年度引用




书目名称Neurodegenerative Diseases年度引用学科排名




书目名称Neurodegenerative Diseases读者反馈




书目名称Neurodegenerative Diseases读者反馈学科排名

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CHAR

Intracellular Signalling in Glutamate Excitotoxicity clear that glutamate-induced activation of N-methyl-D-aspartate (NMDA) receptors for about 5 minutes is sufficient to kill neurons, and that death is expressed within 24 hours of glutamate application. Activation of non-NMDA receptors by, for example, kainate requires exposures of more than 30 minutes; death ensues over a similar time frame.

Goblet-Cells

LAITY

Lymphocyte Amyloid Precursor Protein mRNA Isoforms in Normal Aging and Alzheimer’s Diseased neurons.. Isoform changes have also been observed in AD and following astrocyte, glial and microglial, cell activation.. It is still unknown which isoforms of APP and which cell types contribute to β/A4 peptide deposition in AD although a number of different cell types are potential contributors to the amyloid load.

Dawdle

HIV-1 Coat Protein GP120 Induces Neuronal Injury to Cultured Dopamine Cellsoncentrations (pM) in various neuronal culture systems. as well as modify astrocyte function.. The mechanism(s) by which gp 120 induces neurotoxicity are not understood, but there is evidence that NMDA receptors, Ca. channels, and nitric oxide (NO) are important mediators..

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