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Titlebook: Neuro-Ophthalmology; Global Trends in Dia Andrew G. Lee,Alexandra J. Sinclair,Susan P. Molla Book 2019 Springer Nature Switzerland AG 2019

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Venous Stenting for Idiopathic Intracranial Hypertension,nial pressure including papilledema. Management includes weight loss and medications that reduce intracranial pressure, but when these conservative therapies fail, surgical intervention may be warranted to prevent potentially irreversible vision loss. Based upon an observed association between steno
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Imaging of Oculomotor (Third) Cranial Nerve Palsy,lenging in part because of the number of potential neuroimaging choices (e.g., magnetic resonance angiography (MRA), computed tomography angiography (CTA), intra-arterial digital subtraction angiography (DSA), or routine MRI or CT scan). This chapter describes the clinical guidelines in the evaluati
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Traumatic Optic Neuropathy,be injured by direct and indirect trauma. Falls and motor vehicle accidents are the primary causes of TON, and prevalence varies throughout the world, in part because of variable standards for motor vehicle and passenger safety. The precise mechanism for TON has not been established but may be a com
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Workup for Optic Atrophy,cells in the retina, or descending atrophy following destruction of nerve fibers in the optic nerve, chiasm, optic tract and sometimes the retrogeniculate visual pathways. It usually presents with optic nerve pallor that is typically (but not invariably) associated with decreased visual function. Th
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Treatment of Leber Hereditary Optic Neuropathy,l loss. The majority of patients harbour one of three mtDNA point mutations (m.3460G>A, m.11778G>A and m.14484T>C) with the m.11778G>A mutation being the most common cause of LHON worldwide. This mitochondrial optic neuropathy is characterised by the preferential early loss of retinal ganglion cells
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