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Titlebook: Neural and Neuroendocrine Mechanisms in Host Defense and Autoimmunity; C. Jane Welsh,Mary W. Meagher,Esther M. Sternberg Book 2006 Springe

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onian with potential supported on a discrete (finite or infinite) set of points (“sources”). Such point interaction models are (“solvable”) in the sense that their resolvents can be given explicitly in terms of the interaction strengths and the location of the sources. As a consequence the spectrum,
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Stress-induced Changes in Immune Cell Distribution and Trafficking: Implications for Immunoprotectioon of the state of distribution of leukocytes in the body and of the state of activation of the immune system. A stress-induced change in leukocyte distribution within different body compartments is perhaps one of the most underappreciated effects of stress and stress hormones on the immune system.
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The Effects of Restraint Stress on the Neuropathogenesis of Theiler’s Virus-induced Demyelination: Aresponses are initiated by the hypothalamus and translated into action by the hypothalamic-pituitary-adrenal (HPA) axis and the sympathetic nervous system. Products from these two systems (e.g., corticoid hormones and catecholamines) can directly modulate the activity of various immune effector cells (.).
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activity on neural and endocrine functions and behavior. The underlying premise is that the brain and immune system represent a single, integrated system of defense. .978-1-4899-9723-4978-0-387-48334-4
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tivity on neural and endocrine functions and behavior.Includ.Psychoneuroimmunology is the study of interactions between the brain and the immune system. This book provides a clearly written summary of behavioral, neural and endocrine regulation of immune responses and of the effects of immune system
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Introductionetimes symbiotic and sometimes detrimental bidirectional influences of host-on-pathogen and pathogen-on-host responses. In particular, this volume focuses on extraimmune host responses of the central nervous and neuroendocrine systems that are activated during stress and during infection.
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Influenza Viral Infection: Stress-induced Modulation of Innate Resistance and Adaptive Immunitydiers that died during World War I (.). Luckily, there has not been a repeat of the 1918–1919 pandemic. However, recent events such as the emergence of the avian influenza that is currently causing mortality in Asia may signal the evolution of a new, highly virulent influenza virus that might cause a serious worldwide influenza epidemic.
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Social Stress Alters the Severity of an Animal Model of Multiple Sclerosisl dysfunction, and cognitive dysfunction. The etiology of MS remains uncertain; however, considerable evidence suggests that environmental factors interact with genetic factors to cause disease (.; .; .). Suspected environmental factors include viral infection and stress.
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Book 2006l, neural and endocrine regulation of immune responses and of the effects of immune system activity on neural and endocrine functions and behavior. The underlying premise is that the brain and immune system represent a single, integrated system of defense. .
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