Titlebook: Nephrotoxicity; In Vitro to In Vivo P. H. Bach,E. A. Lock Book 1989 Springer Science+Business Media New York 1989 development.etiology.eva

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In Vitro and in Vivo HgCl2-Induced Nephrotoxicity Assessed by Tubular Enzymes Releaseic parameters and electrolytes excretion. However, the acute tubular necrosis involved in many forms of toxic nephropathy is frequently detected and quantified by the evaluation of the urinary activity of marker enzymes form various segments of the nephron. Enzymuria has been shown to be an early an

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Prevention and Reversal of Mercuric Chloride-Induced Increases in Renal Vascular Resistance by Captosed rat kidney (IPRK) allows changes in RVR to be monitored precisely following nephrotoxin exposure. Mercuric chloride causes dose-dependent increases in RVR in filtering perfused rat kidneys (1) . We have examined the prevention and/or reversibility of these alterations in RVR by hydralazine, vera

KIN

Regioselective Acute Tubular Necrosis in Renal Cortical Slices Following HgCl2 and K2Cr2O7: Localizavely, has been reproduced in renal cortical slices (Ruegg et al, 1987a). This demonstrates that the toxicity of these two metals results from the innate sensitivity of these segments of the nephron to the specific metal and is not a result of altered blood flow or feed-back mechanisms . The question

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Early Indicators of Lead Nephropathy1) . Changes in serum creatinine and blood urea nitrogen (BUN) have proved to be relatively insensitive indices of renal impairment in individual workers, although epidemiological studies in large numbers of workers have indicated a statistically significant increase in both parameters with length o

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Hormones

Bodil Schmidt-Nielsenry systems, especially multiscale modeling and coupled simulations. The cardiovascular and respiratory systems are tightly coupled, as their primary function is to supply oxygen to and remove carbon dioxide from the body‘s cells. Because physiological conduits have deformable and reactive walls, mac

Ingredient

R. A. Goyer,C. R. Weinberg,W. M. Victery,C. R. Millerry systems, especially multiscale modeling and coupled simulations. The cardiovascular and respiratory systems are tightly coupled, as their primary function is to supply oxygen to and remove carbon dioxide from the body‘s cells. Because physiological conduits have deformable and reactive walls, mac

GRE

Gary L. Diamond,Thomas W. Clarkson,John B. Hursh,M. George Cherianry systems, especially multiscale modeling and coupled simulations. The cardiovascular and respiratory systems are tightly coupled, as their primary function is to supply oxygen to and remove carbon dioxide from the body‘s cells. Because physiological conduits have deformable and reactive walls, mac

Fillet,Filet

Z. H. Endre,L. G. Nicholls,P. J. Ratcliffe,J. G. G. Ledinghamh with practical clinical applications.Presents a consistent.Signal transduction pathways are at the core of most biological processes and are critical regulators of heart physiology and pathophysiology.  The heart is both a transmitter and dynamic receptor of a variety of intracellular and extracel