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Titlebook: Navigating the Technological Tide: The Evolution and Challenges of Business Model Innovation; Proceedings of the I Bahaaeddin Alareeni,Alla

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楼主: Hypothesis
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Rashmi Mishra,Deepika Varshney,Jawaher Albalushi,Rawan Albalushi,Mariya Aljaradinsmitter which has probably most captured the imagination of laymen and academics alike – is as elusive as the clinical aspects of serotonergic medications. Why are drugs acting at certain serotonin receptors efficacious against generalized anxiety disorder, but not panic disorder? Why is the invers
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Michael Christian,Yudhiet Fajar Dewantara,Henilia Yulita,Sunarno Sunarno,Kurnadi Gularso waking (W) and to inhibit (permissive role) REM sleep (REMS). Serotonergic neurons of the dorsal raphe nucleus (DRN) fire at a steady rate during W, decrease their firing during slow-wave sleep (SWS), and virtually cease activity during REMS. Serotonin released during W activates 5-HT. somatodendri
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Gurusha Mengi,Sanah Mengiwever, no published neurochemical dream model invokes serotonin as a dream-promoting neuromodulator or accounts for serotonergic dream enhancement. An experimental study of normal volunteers showed that, although SSRI treatment decreased dream recall frequency, several subject-rated dream-intensity
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Imen Ben Slimene,Nadia Mansourord Barry Jacobs once memorably commented that the problem in understanding the role of serotonin in the brain was that it was implicated in virtually eve- thing but responsible for nothing [1]. Serotonin (5-HT) is defnitely implicated in the regulation of sleep but what does it do? Once seen as a s
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Mohammad Faraz Naim,Meera Peethambaran to energy balance. Localized uniquely to neurons with cell bodies in the lateral hypothalamus, orexin has widespread projections throughout the neuraxis, including the raphe nuclei. Dense orexinergic innervation has been noted in the dorsal raphe, where direct axodendritic contacts between orexiner
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