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Titlebook: Natural Resistance Mechanisms of Plants to Viruses; Gad Loebenstein,John Peter Carr Book 2006 Springer Science+Business Media B.V. 2006 Br

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楼主: Stenosis
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Viral Counter-Defense Moleculese in late 1998, with the publication of three seminal papers (.; .; .). These studies demonstrated that specific viral-encoded proteins, shown to enhance pathogenicity when expressed from viral vectors, could suppress the silencing of a reporter transgene. A fourth paper also published in 1998, demo
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Dark Greens Islands: the Phenomenonnck identified the casual agent of tobacco mosaic disease as a contagious solution a “.” and described dark green blotches on the upper leaves of infected plants (.). When a mosaic virus infects a plant, these discrete regions of dark green tissue occur only on leaves that are systemically infected
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Reducing Virus Associated Crop Loss Through Resistance to Insect Vectorscts attack crops, causing damage and reducing yields and crop quality. Insects cause crop loss directly through feeding on leaves, flowers, fruit or seed. A subset of insects damages crops indirectly, through transmission of plant viruses, resulting in reduced yield and crop quality. Breeding for di
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Cross-Protection. Eighty years have passed since the phenomenon was first demonstrated by McKinney (1929), who observed that in tobacco plants systemically infected with a “light green strain” of . (TMV: Genus .), the appearance of yellow symptoms after re inoculation with a TMV “yellow mosaic strain” was repressed
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Arrest in Viral Transport as the Basis for Plant Resistance to Infectionement occurs through intercellular connections, plasmodesmata (PD), between epidermal (EP) cells and mesophyll (MS) cells, or MS cells and MS cells. Systemic movement is more complex, comprising three distinct stages: viral entry into vascular system from MS cells in the inoculated leaf, long distan
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