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Titlebook: Nasser at War; Arab Images of the E Laura M. James Book 2006 Palgrave Macmillan, a division of Macmillan Publishers Limited 2006 battle.bib

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书目名称Nasser at War
副标题Arab Images of the E
编辑Laura M. James
视频video
图书封面Titlebook: Nasser at War; Arab Images of the E Laura M. James Book 2006 Palgrave Macmillan, a division of Macmillan Publishers Limited 2006 battle.bib
描述From his 1956 Suez triumph to the 1967 defeat, President Nasser of Egypt dominated the Arab revolution. Drawing on new Arabic material, this history casts a fresh light on Nasser‘s era and legacy of conflict and provides an essential background to developments in the contemporary Arab world.
出版日期Book 2006
关键词battle; bibliography; conflict; development; imperialism; intervention; revolution; war
版次1
doihttps://doi.org/10.1057/9780230626379
isbn_softcover978-1-349-28224-1
isbn_ebook978-0-230-62637-9
copyrightPalgrave Macmillan, a division of Macmillan Publishers Limited 2006
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A Very Big Forest: Lions and Tigers and Bears,ments came home to be welcomed as victorious heroes — and quietly replaced by others.. By the first anniversary of the revolution, the Egyptian Expeditionary Force in Yemen numbered about 30,000 (nearly one third of Nasser’s entire army), and had been re-organised for a long-term occupational role, holding key towns and lines of communication..
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Eliminate the Past: Nationalising the Suez Canal Company,ting (relatively prompt by Cairo standards), they were roundly scolded. When they were shown in to see the President, his opening gambit was equally disconcerting. ‘What do you know about the Suez Canal?’ he asked.
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pathological myelin formation. In demyelinating CMT, Schwann cell functions are primarily impaired. Various causative mutant proteins are perceived to result in impairment of fundamental cell function during myelination and maintenance; myelin production, degradation of myelin protein (physiologica
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Laura M. James constitute clinically relevant mechanical and toxic models of axonal degeneration. In addition, we describe three different methods to evaluate axonal regeneration using quantitative methods. These protocols constitute a valuable tool to analyze in vitro mechanisms associated to axonal degeneration
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