Titlebook: NF-κB-Related Genetic Diseases; Gilles Courtois,Alessandra Pescatore,Anna Senegas Book 2016 The Author(s) 2016 Genetic diseases.Immunodefi

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Gilles Courtois,Alessandra Pescatore,Jérémie Gautheron,Francesca Fusco,Matilde Valeria Ursini,Anna Susive, and sustainable, and results in sustained growth of farmers’ incomes. The book, instead of looking for global best practices and evaluating them to assess the possibility of replicating these domestically, looks inward at the best practices and experiences within Indian states, to answer ques

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,The NF-κB Signaling Pathway: Players and Functions,t modulating the expression of hundreds of genes that regulate key physiological processes such as immunity, inflammation, cell death, and cell proliferation. Located in the cytoplasm, NF-κB proteins can be activated by a plethora of stimuli in a large variety of cell types. Two pathways of NF-κB ac

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,Genetic Diseases Affecting Ubiquitination Processes in NF-κB Signaling,ly, modifications by K63- and linear-linked polyubiquitin chains allow the recruitment and activation of essential kinases such as TAK1 and IKK1/IKK2. Human pathologies linked to impaired ubiquitination were recently identified with mutations affecting LUBAC, the main E3 complex catalyzing linear ch

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,Lessons Learned from Studying NF-κB-Related Genetic Diseases,the bones, and the central nervous system (CNS). On one side, this allows to compare these abnormalities with those observed in mouse strains affected for the same components and, on the other, to assign specific defects in humans to discrete molecular impairments. All these information should contr

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Book 2016ted pathologies affecting the components of the NF-KB signaling pathway offers the opportunity to understand the various functions of NF-KB in humans, complementing studies performed with mouse models. In addition, people treating those patients acquire a deeper understanding of the molecular basis of the pathophysiological processes..

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https://doi.org/10.1007/978-3-319-25850-8Genetic diseases; Immunodeficiencies; NF-kB signaling pathway; Signal transduction; Skin diseases