Titlebook: NASH and Nutritional Therapy; Kiwamu Okita (Professor and Chairman) Conference proceedings 2005 Springer-Verlag Tokyo 2005 Hepatitis.Hepat

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Kazuyoshi Yonezawam through the soil (Arshad et al. 2008) or via the atmosphere (Uzu et al. 2010). Among common pollutants that affect plants, lead is among the most toxic and frequently encountered (Cecchi et al. 2008; Grover et al. 2010; Shahid et al. 2011). Lead continues to be used widely in many industrial proce

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Nonalcoholic Fatty Liver (NAFL): Overview,vere condition often associated with obesity, type 2 diabetes, and hyperlipidemia. The spectrum of disorders which fall under the term ‘NAFL’ include simple steatosis, steatosis with mild inflammation, and steatosis with inflammation and varying degrees of fibrosis. Most investigators reserve the te

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Genetic Background of Japanese Patients with Nonalcoholic Steatohepatitis (NASH),genesis. Two functional polymorphisms were studied: the −493 G/T polymorphism in the promoter of microsomal triglyceride transfer protein (MTP) and the 1183 T/C polymorphism in the mitochondrial targeting sequence of manganese superoxide dismutase (MnSOD). The G allele in the MTP promoter leads to d

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Predictors of Nonalcoholic Steatohepatitis in Japanese Patients: Thioredoxin and NASH,y the clinicopathological features of NASH, we analyzed patients with NASH and simple fatty liver (FL). Seventy-five patients with biopsy-proven NASH and 53 patients with biopsy-proven FL were enrolled. Their clinical characteristics and histological findings were compared. Detection of GB virus (GB

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Clinical Features of Patients with Nonalcoholic Fatty Liver Disease,and ninety-three patients were diagnosed as having NAFLD at Tokyo Women’s Medical University or an affiliated hospital, from 1990 to September 2003, and their clinical data were collected prospectively. The diagnosis of NAFLD was based on the following criteria: (1) the presence of steatosis (affect

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Mammalian Target of Rapamycin (mTOR),y that regulates cell growth and proliferation. The activity of mTOR is controlled by amino acids (especially by leucine, one of the branched-chain amino acids), in addition to growth factors and overall energy supply through an AMP-activated protein kinase. mTOR, in complex with two other proteins,