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Titlebook: Microcirculation; Blood-Vessel Interac John Grayson,Walter Zingg Book 1976 Plenum Press, New York 1976 Pet.circulation.growth.life sciences

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Blood Vessel Structureunts of membrane, the fusion of vesicles to the plasma membrane, its breakdown and fate, and the energy required for these processes. These difficulties may be avoided by assuming the existence of an intracellular tubular system which could be responsible for bulk transport across the cell by peristaltic action.
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Pulmonary Circulationan be related to macroscopic differences in the topography of the larger vessels. In amphibians, the large arteries remain as far as possible under the pleura, and the large veins are situated toward the interior of the lung; this is central differentiation.
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LymphaticsWe (Tsuchiya .., 1973) have reported that the lymphangiectasia of the small intestine was revealed not only in protein-losing enteropathy but also in liver cirrhosis and Behçet’s disease. Although lymphangiectasia of the small intestine does not necessarily cause enteric protein loss, what mechanism does trigger this loss?
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Methods for the Investigation of the Microcirculationa gasket when inflated and a fingertip pneumoplethysmograph. The compliance is obtained from the mean amplitude of the volume pulse waves . under the effect of the cuff pressures (P . and P .) by . during the dicrotic phase defined in this study. The value C. (= C.) obtained from the data during the
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Red Cell Interactions with the Microcirculation 320× and 400× optical magnification over an averaging time period of 10 ms. . profiles are uniformly nonsymmetrical, the RBCs exhibit rotation, and they frequently deviate sidewise from the overall axial direction of motion. In general, this is more pronounced on the venous side. Since all of the p
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