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Titlebook: Microbial Toxins in Foods and Feeds; Cellular and Molecul Albert E. Pohland,Vulus R. Dowell,Morris E. Potter Book 1990 Plenum Press, New Yo

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Rapid Method for Detection of Pathogenic , and , Using Enzyme Immunoassaygainst virulent plasmid-coded specific proteins of . serotype 03. Then, EIA was performed with the antiserum and proteins of various . strains of different origin. Positive reaction in EIA was observed when the proteins of virulent plasmid-bearing strains were tested. These strains were Y. enterocol
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Oligodeoxyribonucleotide Probe Specific For The 230 Kilobase Pair Virulence Plasmid in Enteroinvasivs are due to the ingestion of either contaminated water or, as is now becoming increasingly apparent, foods. The infective dose for humans is 10. to 10. bacterial cells, with an incubation period of 12–50 hours. The illness is characterized by cramps, nausea, diarrhea and bloody stools and usually l
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Usefulness in the Epidemiology of Food Poisoning Cases of Detection of Specific Restriction Endonuclstriction endonucleases with different specificities has been reported.. Restriction endonucleases have been screened in this laboratory for pathogenic bacteria belonging to the . in the hope that: (i) the detection of specific restriction endonuclease is found at a high frequency in the species; an
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Genetics of Thermostable Direct Hemolysin of ,n important agent of gastroenteritis in other parts of the world.. Although many possible virulence factors have been suggested to explain the pathogenic mechanism of the organism, the thermostable direct hemolysin (TDH) has been considered the major virulence factor based on epidemiological finding
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Genetic Aspects of ,roduced by types C and D, respectively, were shown to be mediated by specific bacteriophages. Evidence is presented that strongly suggests that both neurotoxin and bacteriocin production by type G are in some manner related to a 81-MDa plasmid carried by toxigenic strains..Antigenicity and host rang
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Molecular Structure of , Progenitor Toxinsf a toxic and a nontoxic component (Fig. 1). The molecule of the progenitor toxin dissociates when exposed to pH 7.2 or higher. The dissociated toxic component was named “derivative toxin.”. The derivative toxin is the true “neurotoxin,” but it does not seem entitled to be called “botulinum toxin,”
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