Titlebook: Meßentladungsstrecken (Ionenstrecken); Siegfried Franck Book 1931 Julius Springer in Berlin 1931 Energie.Felder.Funkenstrecke.Gasentladung

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书目名称Meßentladungsstrecken (Ionenstrecken)影响因子(影响力)




书目名称Meßentladungsstrecken (Ionenstrecken)影响因子(影响力)学科排名




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书目名称Meßentladungsstrecken (Ionenstrecken)网络公开度学科排名




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书目名称Meßentladungsstrecken (Ionenstrecken)被引频次学科排名




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书目名称Meßentladungsstrecken (Ionenstrecken)读者反馈学科排名

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expressed and regulated only in estrogen-responsive cell lines, whereas the majority were expressed in all the cell lines tested but regulated only in those that were estrogen responsvie. Sequencing of the cDNA clones established that one mRNA (pNR—100) encoded cathepsin D and that another (pNR—2) c

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Siegfried Franckexpressed and regulated only in estrogen-responsive cell lines, whereas the majority were expressed in all the cell lines tested but regulated only in those that were estrogen responsvie. Sequencing of the cDNA clones established that one mRNA (pNR—100) encoded cathepsin D and that another (pNR—2) c

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yma and Keese, 1992). Plants, apart from creating physical and mechanical barriers, rely mostly on chemical protection against insect feeding. Numerous terpenoid, alkaloid, and phenolic feeding inhibitors have evolved in the course of this arms race between plants and insects (Harborne, 1993; Rosent

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n of the RNA over a limited range of concentrations of tamoxifen. The pNR—2 mRNA was measured in breast tumors. The pNR—2 expression was only detected in tumors expressing estrogen receptor mRNA, and pNR—2 expression was associated with response to tamoxifen in a group of patients receiving primary

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Siegfried Franckn of the RNA over a limited range of concentrations of tamoxifen. The pNR—2 mRNA was measured in breast tumors. The pNR—2 expression was only detected in tumors expressing estrogen receptor mRNA, and pNR—2 expression was associated with response to tamoxifen in a group of patients receiving primary

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Siegfried Franckn of chromosome 11, but not chromosome X nor 13, suppresses the tumorigenic phenotype of a Wilm’s tumor cell line, indicating the tumor suppressor gene for pediatric nephroblastoma is located on chromosome 11 [7]. These genes are actively expressed in normal cells and are mutationally inactivated in

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