Titlebook: Metabolic Changes Induced by Alcohol; G. A. Martini,Ch. Bode Book 1971 Springer-Verlag Berlin · Heidelberg 1971 cancer.central nervous sys

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Influence of Ethanol on the Metabolism of the Protein-deficient Fatty Liver to acetaldehyde. This reaction is catalyzed by the enzyme alcohol dehydrogenase, which contains zinc; it is mainly located in the liver cytoplasm (11). Nicotin-amide adenine dinucleotide, NAD, functions as the coenzyme and hydrogen acceptor (Fig. 1).

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On the Independence of the Ethanol-Induced Triglyceride Accumulation in the Liver from Metabolic Chased fatty acid synthesis as the main source of the lipid accumulation (8,9). This stimulation of fatty acid synthesis in the liver following ethanol administration is considered to be caused by an increased production of hydrogen and acetate (acetyl-CoA) during ethanol oxidation. On the other hand,

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Effects of Ethanol on Gluconeogenesisr responsible for the hypoglycaemia observed when alcohol is consumed in the fasting or semi-fasting state (1). The reasons why alcohol inhibits hepatic gluconeogenesis are at least partially understood: the facts agree with the assumption that the primary process is the interaction between ethanol

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H. Goebell,Ch. Boderol, in particular with regard to the interpretation and evaluation of emotions. Analysed with a modern as well as a specifically medieval concept of emotion in mind, in its emphasis on action and interaction the normative genre of the ›courtesy book‹ is crucial for the development of a late medieva