Titlebook: Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma; Alexander Baethmann,Oliver S. Kempski,Frank Staub Conference proceed

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Apoptosis in Focal Cerebral Ischemia evidence of apoptosis in the rodent (rat, mouse) brain after middle cerebral artery occlusion. Emphasis will be placed on describing the temporal profile and the anatomical distribution of cells undergoing apoptosis as functions of duration of MCA occlusion and reperfusion after MCA occlusion. The

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ironic

Three-Dimensional Metabolic and Hemodynamic Imaging of the Normal and Ischemic Rat Brain Fourier analysis revealed satisfactory retention of low-frequency information. The method was then applied to study metabolism/blood flow relationships in the acute focal ischemic penumbra of Sprague-Dawley rats subjected to distal photothrombotic middle cerebral artery (MCA) occlusion, coupled wit

FUME

Origins of Glutamate Release in Ischaemiandition..These results, and other considerations, do not favour the view that presynaptic glutamate release and reversed glutamate uptake are suitable targets for neuroprotection in ischaemia. Acting post-synaptically to inhibit recurrent spreading depression (NMDA-receptor antagonists) or to modula

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Thrombolysis in Acute Ischemic Strokeal artery, were excluded from the study..Other ongoing trials on thrombolytic agents are expected to provide further indications on how to identify those patients most likely to benefit and least likely to experience adverse effects from this treatment.

SOB

Traumatically Induced Axonal Damage: Evidence for Enduring Changes in Axolemmal Permeability with Asng altered axolemmal permeability to the peroxidase were assessed at the light and electron microscopic level. Through this approach, we recognized that a traumatic episode of moderate severity evoked changes in axolemmal permeability which surprisingly endured for up to 5 hrs postinjury. At such fo