Titlebook: Maturation Phenomenon in Cerebral Ischemia; Proceedings of the S Umeo Ito,T. Kirino,Igor Klatzo Conference proceedings 1992 Springer-Verlag

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Simultaneous Mapping of Superoxide Free Radicals, Vascular Permeability, and Energy Metabolism in Fex, and dorsal hypothalamus and by increasing at a later stage in the CA3..In severe postischemic lesions, the critical level of free radicals which caused vasogenic edema with tissue ATP crisis was 2.5 times higher than the normal values..As sources of these free radicals, arachidonic acid release

IDEAS

Carotid Artery Back Pressure and Postischemic Hyperthermia in the Gerbil: Factors Influencing the Dneurons from 24% to 89% of the controls (P < 0.05). However, the protective effect was lost when anesthetized animals were brought to the same hyperthermic temperature profile as the nonanesthetized ones..These observations demonstrate that the density of ischemia and the postischemic temperature pr

Ornament

Maturation of Ischemic Injuries Observed in Mongolian Gerbils: Introductory Remarks, those selected for study showed more than 10 points on the stroke index score following clipping of the left common carotid artery [17, 25]. Following less than 1-h temporary ischemia, the computer-analyzed EEG recovered rapidly during the first l-h and deteriorated again at 48 h after restoration

Nmda-Receptor

虚情假意

Reorganization in the Gerbil Hippocampus After Ischemia-Induced Delayed Neuronal Death: Fate of Par pyramidal cells degenerate; GABAergic neurons are resistant against the factor(s) causing ischemicirreversible nerve cell damage. A subpopulation of these GABA ergic neurons containsthe calcium-binding protein parvalbumin, suggesting that the calcium-binding property of single cell types is one, bu

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Changes in Hippocampal Ultrastructure After Ischemia with Long Survival Times,e studied. After 3 weeks of recirculation, the number of pyramidal neurons in the CA1 sector had decreased to about 6%, a value which did not further change after 6 or 10 months survival. Neuronal death was followed by a marked shrinkage of all CA1 layers becoming obvious after 6 months. Electron mi

Agility

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Reversibility of Damage to Rat Cerebellar Purkinje Cells Following Ischemia,ay, 2 days, or 4 days following the ischemic insult. Ischemic neuronal alteration was always found in the cerebellum throughout the observation period. Three hours following ischemia, Purkinje cells looked darkly stained to basic dyes such as toluidine blue and hematoxylin. Six hours after ischemia,

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Neuropathological Changes in Remote Areas After Focal Cerebral Ischemia,a nigra, after focal cerebral ischemia. In the rat middle cerebral artery occlusion model, the ipsilateral thalamus progressively shrank in the course of several months. The thalamic atrophy is most likely due to disconnection of neuronal fibers between the cerebral cortex and the thalamus. In the i