Titlebook: Mathematical Demography; Selected Papers David P. Smith,Nathan Keyfitz Textbook 19771st edition Springer-Verlag Berlin · Heidelberg 1977 Ca

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Edmund Halleye could not be infected because they lacked expression of the protein that was now 3,4 forever linked to these disease . Since then it has become more widely accepted for these diseases to be termed prion disea978-1-4899-8688-7978-0-387-23923-1

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George Kinge could not be infected because they lacked expression of the protein that was now 3,4 forever linked to these disease . Since then it has become more widely accepted for these diseases to be termed prion disea978-1-4899-8688-7978-0-387-23923-1

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Edward S. Deevey Jr.e could not be infected because they lacked expression of the protein that was now 3,4 forever linked to these disease . Since then it has become more widely accepted for these diseases to be termed prion disea978-1-4899-8688-7978-0-387-23923-1

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John Grauntmutant PrP. are believed to result in the neurotoxicity ob-served in familial cases of prion disorders.. However, prion-specific neuropathology is often observed in the absence of detectable PrP., indicating the presence of alternative pathways of neurotoxicity incertain cases of prion disorders.. R

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Lowell J. Reed,Margaret Merrellmutant PrP. are believed to result in the neurotoxicity ob-served in familial cases of prion disorders.. However, prion-specific neuropathology is often observed in the absence of detectable PrP., indicating the presence of alternative pathways of neurotoxicity incertain cases of prion disorders.. R

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