Titlebook: Management of Patients with Pseudo-Endocrine Disorders; A Case-Based Pocket Michael T. McDermott Book 2019 Springer Nature Switzerland AG

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Bewildered by Biotin,and nails. While the daily requirement for biotin is around 30–70 mcg, most supplements contain high doses of 5000–20,000 mcg (5–20 mg). These supplements result in blood levels of biotin that can interfere with common laboratory assays, specifically those using biotin–streptavidin chemistry. In the

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Idiopathic Postprandial Syndrome, et al. Diabetes Metab 26:337–351). This terminology is purely a description of the timing of a hypoglycemic event; an evaluation for a possible etiology should subsequently be pursued. Patients with hypoglycemic symptoms but without chemical hypoglycemia are often erroneously characterized as havin

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,Adrenal Insufficiency, “Relative Adrenal Insufficiency,” or None of the Above?,emely challenging and complex. It is suggested that serum total cortisol levels and the response of total cortisol to a cosyntropin stimulation test are not reliable markers of adrenal function in severe illness due to multiple factors, including hypoalbuminemia, low serum cortisol binding globulin

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,Pseudo-Cushing’s Syndrome: A Diagnostic Dilemma,gic or non-neoplastic causes. A thorough history including concurrent medications and medical problems is vital. The most common causes of non-neoplastic physiological hypercortisolism, often referred to as pseudo-Cushing’s syndrome, are alcoholism and alcohol withdrawal, chronic kidney disease, dep

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,Pseudo-Cushing’s Syndrome: Alcohol Abuse, Obesity, and Psychiatric Disorders,iatric diseases. Distinguishing physiologic from pathological/neoplastic causes of Cushing’s syndrome is challenging because of the potential overlap in clinical presentations and screening test abnormalities.