Titlebook: Malnutrition in the Elderly; W. O. Seiler,H. B. Stähelin Conference proceedings 1999 Dr. Dietrich Steinkopff Verlag GmbH & Co. KG, Darmsta

江湖郎中

Nonporous

Dictation

http://image.papertrans.cn/m/image/622000.jpg

jaundiced

Introductiontly to morbidity and mortality in the aged. The immune function is impaired, the risk for falls and fractures increases, in acute illness, recovery is delayed, and complications are frequent. Acute and chronic illnesses lead to a catabolic metabolism and hence increase the signs and symptoms of maln

亵渎

Nutrient intake of healthy elderly subjects. At the age of 74–79 years subjective health was satisfactory or good in 95% of 399 men and 93% of 414 women in six study towns. In these subjects suboptimal blood levels of nutrients were virtually nonexistent. Food intake was low in energy and rich in protein and fat. Lowest European recommended

掺假

Nutritional status in ill elderly subjectsDepending on the type and composition of the group of patients under consideration, the prevalence of malnutrition is cited at up to 60%. The elderly eat considerably smaller amounts of food than the young and mostly eat food of low nutrient density. Especially at times of high energy requirements s

组成

Obvious

Aging and water metabolism in health and illnessintenance of water and sodium balance. The presence of many diseases and drugs common in the elderly can interact with the impaired homeostatic systems to result in clinically significant disturbances of water and sodium with accompanying symptoms, morbidity, and mortality. These disorders, which in

钢笔尖

Malnutrition and mental functionsse deficits. Malnutrition impairs cognitive performance only when micronutrient levels fall below certain critical thresholds. Malnutrition in the elderly is commonly the result of preexisting mental impairment due to chronic diseases. These also lead to accelerated neuronal destruction via a stress

concubine

Undernutrition and osteoporosis strength. Furthermore, the rate of medical complications after fracture can also be increased by nutritional deficiency. The IGF-I system appears to be directly involved in the pathogenetic mechanisms leading to osteoporotic hip fracture in the elderly and to its complications. In the presence of a