Titlebook: Malignant Transformation by Viruses; W. H. Kirsten Book 1966 Springer Science+Business Media New York 1966 Viruses.cancer.genetics.growth.

Charlatan

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疯狂

Discussionenome with the host cells. This we can state with unquestionable certainty. Whether their presence in virus-free cells indicates persistence of the viral genome or a fraction of it, we cannot state with equal certainty since attempts to obtain independent proofs of viral genome integration in the ca

ETHER

Complementation Between Defective Oncogenic Virusesequires complementation for replication is usually defective in some portion of its genome. However, some viruses competent to grow unilaterally in one cell may be incompetent in a second cell and thus require a second virus to furnish help for some step in the replicative cycle. Though complementat

LUMEN

Cytogenetic Alterations During Malignant Transformationly relates to various theories of cancer which can be assembled under the term “somatic mutation.” Only in the past 7 or 8 years have techniques been available for adequate studies of mammalian chromosomes. The comments of Schultz (18) are appropriate in this regard: “It is a truism by now that the

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Transformation of Human Cells by Oncogenic Virus SV40virus type 7-SV40 (strain LLE-46) (4). This latter virus is a human strain of adenovirus which has apparently acquired a portion of the SV40 genome during laboratory passage. By currently accepted standards, infection of human tissue with Rous sarcoma or wart viruses has not led to transformation.

Hyperalgesia

Immunologic Attack on Neoplasiahich initiate the neoplastic process and second, protecting the host against the invaders from within which are the cancer cells themselves. The immunologic approach affords the means for attack both upon the invaders from without and the invaders from within. For convenience the immunologic approac

Deadpan

Round Table Discussionse investigators, when they talk about the same things, to use the same words. I think this would help the audience. If I was sitting in the audience right now, I would applaud that suggestion! It is hard enough to follow some of the very involved experiments that have been described today, without

Archipelago

Discussionisappear in the adult, to reappear again when a neoplastic process has occurred. One is the case of the antigen in rat liver hepatomas (1), the other is the antigen observed in colon carcinomas of man (7). These are therefore examples in which the expression of specific cellular antigens is modulate