Titlebook: Lymphohaematopoietic Growth Factors in Cancer Therapy II; Roland Mertelsmann Conference proceedings 1992 Springer-Verlag Berlin Heidelberg

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ESO Monographshttp://image.papertrans.cn/l/image/589218.jpg

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Introduction and Overviewmay be cloned or expanded . in order to perform adoptive transfer strategies, they can be stimulated to proliferate and to exert specific functions by employing recombinant cytokines or by transfection of optional genes. Under . conditions “physiological” mechanisms can be specifically manipulated b

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Pathophysiology of T-Cell Mediated Shock Induced by Bacterial Superantigensitive organisms cause this clinical syndrome. Various aspects of the pathophysiology of gram-negative bacteraemia-induced septic shock are increasingly understood. For example endotoxin, an LPS component of the gram-negative outer membrane, causes cells of the macrophage lineage to produce cytokines

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Natural Killer Cells and Interleukin-2-Activated Killer Cellsy was described some years ago as “natural cytotoxicity” because it was present spontaneously in normal individuals and was independent of histocompatibility complex (MHC) [1,2]. The principal effectors of natural cytotoxicity have been identified morphologically as large granular lymphocytes (LGL)

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Tumour Immunogenicity Induced by Exogenous Interleukinsells [1–4] and the ephemerality of that elicited during tumour growth is a paradox around which research in tumour immunology has danced for about a hundred years. The rationale behind both old and new attempts at immunotherapy returns to similar issues in an almost circular loop reflecting the pers

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Analysis of T-Cell Receptor Variability in Tumour Infiltrating Lymphocytes based on the . infusion of cells displaying . antitumour cytotoxic activity. This approach is most often combined with host system stimulation (“active immunotherapy”) aimed at the rejection of the tumour by, for example, infusions of IL-2, a pivotal mediator of the immune system. In animal models,

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