Titlebook: Lung Cancer; Advances in Basic an Heine H. Hansen Book 1995 Springer Science+Business Media New York 1995 Staging.cancer treatment.carcinom

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Smoking cessation programs,g the last decade, and today we have well-developed, relatively effective treatment modalities for smoking cessation. The cost of smoking cessation (i.e., cost per saved year of life) is several times cheaper than treatment of comparable risk factors such as hypertension and hypercholesterolemia.

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Neuroendocrine characteristics in malignant lung tumors: Implications for diagnosis, treatment, ande Precurser Uptake and Decarboxylation) because of their amine-handling properties, one of their main functions being their ability to take up and decarboxylate relatively simple amino precursor substances, such as dihydroxyphenylanaline (DOPA) and 5-hydroxytryptamine (5-HT), thus producing biogenic

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In vitro models for testing of cytostatic agents in small cell lung cancer,rug resistance is the major problem; only a small proportion of patients will remain in remission beyond two years [1]. The overall treatment results have not changed markedly in the last decade, despite the application of numerous combination regimens, late intensification regimens, high-dose chemo

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Multidrug resistance in lung cancer,y referred to as non-small cell lung cancers (NSCLC), and a distinct type of lung cancer, small cell lung cancer (SCLC) [1]. Surgery, radiotherapy, and chemotherapy are used for the treatment of lung cancer, but long-term survival is obtained in only 5% to 10% of the patients. Thus, improvements in

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New techniques in the diagnosis and staging of lung cancer,ally (without performing unnecessary invasive procedures), and pragmatically (without incurring extra cost). The end result should not rest on inferences of interpretation, but must be established beyond doubt on the basis of pathological material. There is a close relationship between pathological

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Interferon and lung cancer,e types of interferon (IFN): .(IFNα), produced by leukocytes; . (IFNβ), produced by fibroblasts, macrophages and epithelial cells; and .(IFNγ), produced by activated T-lymphocytes and natural killer lymphocytes. There are at least 17 genes controlling the production of IFNα, but only one gene for IF