Titlebook: Lipoproteins in Diabetes Mellitus; Alicia J. Jenkins,Peter P. Toth Book 2023Latest edition The Editor(s) (if applicable) and The Author(s)

荧光

The PPAR System in Diabetes in inflammation. binding to specific response elements (PPREs) within the nucleus. Heterodimerization with a retinoid X receptor and interaction with coactivators and corepressors occur after binding of natural or synthetic agonists. The first synthetic agonists in clinical use were fibrates for PP

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Exposition

Triglyceride- and Cholesterol-Rich Remnant Lipoproteins in Risk of Cardiovascular Disease in Diabetesterol-rich remnant lipoproteins. The cholesterol content of these lipoproteins is most likely atherogenic in the same way as cholesterol held in low-density lipoproteins, according to evidence from epidemiology, Mendelian randomization studies, and clinical trials. In addition, very high levels of

Asymptomatic

HDL Function in Diabeteslin. These disturbances result in impaired cellular glucose transport and hyperglycaemia. The presence of hyperglycaemia is associated with an enhanced inflammatory response and mitochondrial oxidative stress, leading to vascular endothelial dysfunction. Hyperglycaemia is a metabolic feature charact

Cleave

Lipoprotein(a): Metabolism, Pathophysiology, and Impact on Diabetes Mellitussm are slowly beginning to unravel..The concentration of plasma Lp(a) is to >90% inherited where the best studied kringle-4 size polymorphism accounts for >50% of inheritance. Other factors like steroid hormones, dietary fatty acids, and vitamins have minor effects on Lp(a) levels. Secondary factors

Metamorphosis

Lipoprotein Glycation in Diabetes Mellitusd pressure, and blood clotting control, many people with diabetes will develop the macrovascular and microvascular complications of diabetes. The majority of people with diabetes are likely to die of an atherosclerosis related event. Contributing factors to the cardiovascular, renal, retinal, and ne

Engulf

Lipid: Extracellular Matrix Interactions as Therapeutic Targets in the Atherosclerosis of Diabetesrosclerosis in diabetes is contributed to by alternations in both lipoproteins and in the extracellular matrix, and the lipoprotein/matrix interactions. Whilst most studies target circulating lipoproteins, the vascular wall matrix and lipid:extracellular matrix interactions are also potential therap

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The Role of Modified Forms of LDL and Corresponding Autoantibodies in the Development of Complicatio induce immunogenicity. Modified forms of LDL were capable of inducing endothelial dysfunction and vascular inflammation through activation of innate and acquired immunity, thus contributing to the development and progression of diabetic complications. The immunogenicity of the modified forms of LDL

stress-response

Endothelial Dysfunction in Type 2 Diabetes with an Update on New Interventionsetes (T2DM). The pathogenesis of endothelial dysfunction in large and small arteries in T2DM is complex and fundamentally involves uncoupling of endothelial nitric oxide synthase activity, mitochondrial oxidative phosphorylation, and activation of vascular NAD(P)H oxidase, leading to oxidative stres

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