Titlebook: Lipoprotein Deficiency Syndromes; Aubie Angel,Jiri Frohlich Book 1986 The Editor(s) (if applicable) and The Author(s), under exclusive lic

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ELUDE

Clinical Syndrome and Lipid Metabolism in Hereditary Deficiency of Apolipoproteins A-I and C-III, Vat discovery lipid metabolism has been examined in some detail in the two patients with this disorder. This report will review the clinical findings in the two patients and present results of studies on the physico-chemical properties and metabolism of their lipoproteins.

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Genetic High Density Lipoprotein Deficiency States and Atherosclerosisit hepatic chylomicron remnant uptake (16,17). Both apoB and apoE can bind to the apoB,E receptor (LDL receptor) on various cell surfaces, while apoE also binds to the liver apoE receptor, which is essential for chylomicron remnant uptake (18,19). The low density lipoprotein (LDL) receptor appears t

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Metabolic Interrelationships of HDL Subclassesolyaerylamide gradient gels.. Each of these procedures has revealed further heterogeneity within the HDL. and HDL. density subclasses. In the case of gradient gel electrophoresis, heterogeneity is manifest as multiple electrophoretic bands which have been shown to represent distinct HDL subspecies o

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Speciation of HDLof particle populations in plasma must take into account the fact that chemical processes will tend to move toward thermodynamic equilibrium after sampling, thus altering the distribution of species. Approaches to the study of lipoprotein speciation must therefore include the use of techniques which

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Tonometry

Effect of Diabetes Mellitus and End-Stage Renal Disease on HDL MetabolismIn general, patients with this disorder have a decrease of cholesterol and an increase in triglyceride in their HDL. There is an increase in apo E and a decrease in apo CII in their HDL. Apo A-I levels are unaffected whereas apo A-II levels are decreased. Renal transplant patients may have low, norm

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Clinical, Nutritional and Biochemical Consequences of Apolipoprotein B Deficiencyeflect the work in progress in a number of areas, the most important of which is the recognition that the neurological abnormalities seen in ABL are a result of vitamin E deficiency and that these neurological abnormalities can be prevented by the oral administration of vitamin E from infancy and ea

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