Titlebook: Leukemia: Advances in Research and Treatment; Emil J. Freireich,Hagop Kantarjian Book 1993 Kluwer Academic Publishers 1993 cell.cytokines.

left-ventricle

起波澜

Fludarabine phosphate in the treatment of chronic lymphocytic leukemia: Biology, clinical impact, aidentify other clinically active arabinosyl nucleosides. The purine nucleoside analogue, 9-β-D-arabinofurano-syladenine (ara-A), was found to be of little therapeutic value due to its rapid inactivation by adenosine deaminase (ADA) [1]. This metabolic obstacle is overcome, however, by the 2-fluoro d

galley

厌恶

Detection of minimal residual disease in ALL,rmined by the limits of reliable detection by morphology. Mathematical models predict that the level of residual disease during clinical remission could vary from 10. leukemic cells following early remission induction to perhaps none in some patients at later stages of treatment [1]. The practical r

背书

Cytokines and myeloid-specific genes: Patterns of expression and possible role in proliferation andastic precursor cells, both intramedullary and often extramedullary. Other features that also reflect the neoplastic behavior of these cells . include their clonality, the presence of recurrent karyotypic abnormalities, their transplantability, and their often abnormal patterns of expression of and

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痛得哭了

GROWL

寄生虫

The molecular pathogenesis of the philadelphia-positive leukemias: Implications for diagnosis and ty cases of acute lymphoblastic leukemia (ALL) and some cases of acute myelogenous leukemia (AML). In 1973, Rowley demonstrated that the Philadelphia chromosome, a small form of human chromosome 22 [1], was the product of a reciprocal translocation between the long arms of chromosomes 9 and 22: t(9;2

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