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Titlebook: Large Lakes; Ecological Structure Max M. Tilzer,Colette Serruya Book 1990 Springer-Verlag Berlin Heidelberg 1990 Nahrungsnetz.Ocean.Sedimen

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Charles E. Herdendorfin the pathogenesis of .-associated gastroduodenal lesions. Variations in host responses to infection determined by immune response genes may be critical in determining the extent of .-induced mucosal injury. There is also increasing evidence that inflammatory responses may reflect strain variation
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Max M. Tilzerin the pathogenesis of .-associated gastroduodenal lesions. Variations in host responses to infection determined by immune response genes may be critical in determining the extent of .-induced mucosal injury. There is also increasing evidence that inflammatory responses may reflect strain variation
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Dieter M. Imbodenof . infection has resulted in considerable attention being focused on investigations of host-pathogen interactions and the role of cytokines in generation of the chronic inflammatory response.. Cytokines have a major role in regulating the extent and characteristics of both innate and specific muco
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Eckard Hollan,Paul F. Hamblin,Hubert Lehnrease. Sialyl-Lewis x/a antigens (sLex/a) are specifically recognized by the . sialic acid-binding adhesin SabA. Even though bacterial adherence per se cannot cause disease, adherence is considered as a crucial step in pathogenesis since it is needed for bacterial delivery of effector molecules into
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Charles R. Goldman,Alan Jassbyes. Here we review the current status in the characterization of phosphorylation-dependent and phosphorylation-independent signaling events by CagA and the CagA-independent T4SS activities in vivo and in vitro, which include the induction of membrane dynamics, actin-cytoskeletal rearrangements, disr
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