Titlebook: Languages, Applications and Technologies; 4th International Sy José-Luis Sierra-Rodríguez,José-Paulo Leal,Alberto Conference proceedings 20

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Mariana Julião,Jorge Silva,Ana Aguiar,Helena Moniz,Fernando Batistaous types of malignant transformation. Numerous HSP90 inhibitors have been identified so far and many of them have also been clinically tested. Although most of these are natural or their derived inhibitors including geldanamycin and its derivatives 17-AAG and 17-DMAG have shown efficacy, their easy

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Itisree Jena,Himani Chaudhry,Dipti Misra Sharma Sieroter Ital 37:79–108, 1962). Members of this family function as molecular chaperones that stabilize protein folding (De Maio, Shock 11:1–12, 1999). HSP47 is a molecular chaperone that is essential for collagen biosynthesis (Ishida et al., Mol Biol Cell 17:2346–2355, 2006; Matsuoka et al., Mol Bi

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Efthymios Chondrogiannis,Vassiliki Andronikou,Efstathios Karanastasis,Theodora Varvarigous, and its response to treatment, is vital and secreted biomarkers may be useful. One key group of biomarkers are secreted cell stress proteins – now recognised to be potent intercellular signalling molecules. Periodontitis is not an infectious disease, but is driven by oral bacteria and so both bac

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Alberto Simões,José João Almeida throughout the viral lifecycle. The molecular chaperones, heat shock proteins 70 (HSP70) and 90 (HSP90), have been shown to be host factors that are utilized by a wide range of viruses, including HIV, influenza, polioviurs, and dengue virus for replication and propagation. There is an observed incr

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Marcos Garcia,Pablo Gamallo While AD is a widely recognized medical, public health, and social problem, the chaperonopathies have not yet been acknowledged as a related burden of similar magnitude. However, recent evidence collectively indicates that such possibility exists in that AD, or at least some forms of it, may indeed

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y, HSP27 is often increased in cancers and associated with poor patients’ survival and resistance to conventional chemotherapy. Conversely, the role of HSP27 in response to therapies targeted towards oncogenic kinases was poorly characterized. In this chapter we review the findings on the role of HS

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