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Titlebook: Klinische Neuroendokrinologie; Klaus Werder Book 19981st edition Springer-Verlag Berlin Heidelberg 1998 Biologie.Endokrinologie.Hoden.Horm

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Klaus von Werder FRCPls, and primordial germ cells. Homozygotes of the more severe alleles are typically white with black eyes, sterile, and suffer from a severe macrocytic anemia which often results in the death of affected animals in utero, or shortly after birth (Sarvella and Russell, 1956). The effects of . mutation
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Klaus von Werder FRCPls, and primordial germ cells. Homozygotes of the more severe alleles are typically white with black eyes, sterile, and suffer from a severe macrocytic anemia which often results in the death of affected animals in utero, or shortly after birth (Sarvella and Russell, 1956). The effects of . mutation
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Klaus von Werder FRCPing libraries coupled with the increasing popularity in the prospect of sequencing mammalian genomes, has triggered a resurgence of interest in finding and characterizing genes that playa role in modifying immune processes and diseases. Genetically defined strains of mice (e. g. , inbred strains and
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Klaus von Werder FRCPing libraries coupled with the increasing popularity in the prospect of sequencing mammalian genomes, has triggered a resurgence of interest in finding and characterizing genes that playa role in modifying immune processes and diseases. Genetically defined strains of mice (e. g. , inbred strains and
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Klaus von Werder FRCPNA) and the virion contains the enzyme system which transcribes the NA into the viral NA [75]. The synthesis of influenza viral NA involves a unique interaction with the host cell transcriptional machinery in the nucleus of the infected cell. This interaction is required first for the initiation of
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Klaus von Werder FRCPhe hallmark of influenza virus is its variability, having the capacity to change its antigenic identity so remarkably that the specific immunity established in response to infection by a particular strain may give little or no protection against viruses that subsequently arise. Antigenic variation i
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