Titlebook: Islets of Langerhans; Md. Shahidul Islam Reference work 2015Latest edition Springer Science+Business Media Dordrecht 2015 Beta cells.Diabe

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Regulation of Pancreatic Islet Formation,ysfunction of insulin-producing cells (β-cells) causes diabetes. Generation of β-like cells that can compensate the loss of β-cell mass in type 1 diabetes or defects in β-cell insulin secretion in type 2 diabetes is a current challenge in biomedicine. The knowledge of the molecular basis governing p

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(Dys)Regulation of Insulin Secretion by Macronutrients,ional neurohormonal signals, in order to secrete insulin to best meet the needs of the organism. β-Cell nutrient sensing requires metabolic activation, resulting in production of stimulus-secretion coupling signals that promote insulin biosynthesis and release. The primary stimulus for insulin secre

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Physiology and Pathology of the Anomeric Specificity for the Glucose-Induced Secretory Response of mannose. In a physiological perspective, emphasis is placed on the experimental findings documenting such an anomeric specificity and its possible determinants, including consideration on the essential role of the anomeric specificity of phosphoglucoisomerase, phosphomannoisomerase, and phosphogluco

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Electrophysiology of Islet Cells,abolism-dependent alterations in ion channel activity..This coupling is best explored in β cells. The effect of glucose is directly linked to mitochondrial metabolism as the ATP/ADP ratio determines the open probability of ATP-sensitive K. channels .K. channels). Nucleotide sensitivity and concentra

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ATP-Sensitive Potassium Channels in Health and Disease, β-cell is finely balanced; increased activity prevents insulin secretion, whereas reduced activity stimulates insulin release. β-cell metabolism tightly regulates K. channel gating, and if this coupling is perturbed, two distinct disease states can result. Diabetes occurs when the K. channel fails

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